Bone-marrow mesenchymal stem cells reduce rat intestinal ischemia-reperfusion injury, ZO-1 downregulation and tight junction disruption via a TNF-α-regulated mechanism

World J Gastroenterol. 2013 Jun 21;19(23):3583-95. doi: 10.3748/wjg.v19.i23.3583.

Abstract

Aim: To investigate the effect of bone-marrow mesenchymal stem cells (BM MSCs) on the intestinal mucosa barrier in ischemia/reperfusion (I/R) injury.

Methods: BM MSCs were isolated from male Sprague-Dawley rats by density gradient centrifugation, cultured, and analyzed by flow cytometry. I/R injury was induced by occlusion of the superior mesenteric artery for 30 min. Rats were treated with saline, BM MSCs (via intramucosal injection) or tumor necrosis factor (TNF)-α blocking antibodies (via the tail vein). I/R injury was assessed using transmission electron microscopy, hematoxylin and eosin (HE) staining, immunohistochemistry, western blotting and enzyme linked immunosorbent assay.

Results: Intestinal permeability increased, tight junctions (TJs) were disrupted, and zona occludens 1 (ZO-1) was downregulated after I/R injury. BM MSCs reduced intestinal mucosal barrier destruction, ZO-1 downregulation, and TJ disruption. The morphological abnormalities after intestinal I/R injury positively correlated with serum TNF-α levels. Administration of anti-TNF-α IgG or anti-TNF-α receptor 1 antibodies attenuated the intestinal ultrastructural changes, ZO-1 downregulation, and TJ disruption.

Conclusion: Altered serum TNF-α levels play an important role in the ability of BM MSCs to protect against intestinal I/R injury.

Keywords: Bone marrow mesenchymal stem cells; Intestinal mucosa; Ischemia-reperfusion injury; Tumor necrosis factor-α; Zona occludens 1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amine Oxidase (Copper-Containing) / blood
  • Animals
  • Biomarkers / blood
  • Bone Marrow Transplantation*
  • Cells, Cultured
  • Disease Models, Animal
  • Down-Regulation
  • Intestinal Mucosa / metabolism
  • Intestines / blood supply*
  • Intestines / immunology
  • Intestines / ultrastructure
  • Lactic Acid / blood
  • Male
  • Mesenchymal Stem Cell Transplantation*
  • Mesenteric Vascular Occlusion / blood
  • Mesenteric Vascular Occlusion / immunology
  • Mesenteric Vascular Occlusion / pathology
  • Mesenteric Vascular Occlusion / surgery*
  • Mucous Membrane / blood supply*
  • Mucous Membrane / immunology
  • Mucous Membrane / metabolism
  • Mucous Membrane / ultrastructure
  • Permeability
  • Rats
  • Rats, Sprague-Dawley
  • Reperfusion Injury / blood
  • Reperfusion Injury / immunology
  • Reperfusion Injury / pathology
  • Reperfusion Injury / prevention & control*
  • Signal Transduction
  • Tight Junctions / metabolism*
  • Time Factors
  • Tumor Necrosis Factor-alpha / blood*
  • Zonula Occludens-1 Protein / metabolism*

Substances

  • Biomarkers
  • Tjp1 protein, rat
  • Tumor Necrosis Factor-alpha
  • Zonula Occludens-1 Protein
  • Lactic Acid
  • Amine Oxidase (Copper-Containing)