Characteristics of immunosuppressive macrophages induced in spleen cells by Mycobacterium avium complex infections in mice

J Gen Microbiol. 1990 May;136(5):965-73. doi: 10.1099/00221287-136-5-965.

Abstract

The profile of generation and characteristics of splenic macrophages (M phi s) which suppress the concanavalin A (Con A) mitogenic response of splenic T cells (designated as 'immunosuppressive M phi s') in host CBA/JN mice during the course of Mycobacterium avium complex (MAC) infection were investigated. In MAC-infected mice, reductions in some cellular functions of host splenic T cells, such as the Con A mitogenic response and mixed leucocyte reaction, were seen around 2 weeks after challenge of organisms, and this was accompanied by appearance of immunosuppressive M phi s in spleen cells. In this case, increase in immunosuppressive M phi activity was seen in terms of both activity per spleen and activity per individual M phi. In this phase of the infection, MAC-induced splenic M phi s showed a markedly increased ability to produce reactive oxygen radicals in response to phorbol myristate acetate. Thus, the expression of suppressor activity of MAC-induced M phi s seems to be closely linked to their activated state. A large proportion of the immunosuppressive M phi s exhibited suppressor activity dependent on prostaglandins and membrane functions related to microfilaments. It was also found that the generation of IL-2-reactive T cell populations in response to Con A was markedly inhibited by MAC-induced splenic M phi s, whereas they caused no significant reduction in the IL-2-producing ability of normal spleen cells.

MeSH terms

  • Animals
  • Concanavalin A / pharmacology
  • Immune Tolerance
  • Immunity, Cellular
  • Interleukin-2 / biosynthesis
  • Kinetics
  • Lymphocyte Activation
  • Macrophages / immunology*
  • Male
  • Mice
  • Mice, Inbred CBA
  • Mycobacterium avium
  • Spleen / immunology
  • T-Lymphocytes / immunology
  • Tuberculosis / immunology*

Substances

  • Interleukin-2
  • Concanavalin A