Downstream targets of the homeobox gene DLX3 are differentially expressed in the placentae of pregnancies affected by human idiopathic fetal growth restriction

Mol Cell Endocrinol. 2013 Sep 5;377(1-2):75-83. doi: 10.1016/j.mce.2013.06.032. Epub 2013 Jul 2.

Abstract

Human idiopathic fetal growth restriction (FGR) is associated with placental insufficiency. Previously, we reported that the expression of homeobox gene Distal-less 3 (DLX3) is increased in idiopathic FGR placentae and is a regulator of villous trophoblast differentiation. Here, we identify the downstream targets of DLX3 in trophoblast-derived cell lines. We modelled the high levels of DLX3 in FGR using an over-expression plasmid construct and complemented this using short-interference RNA (siRNA) for inactivation in cultured cells. Using a real-time PCR-based gene profiling, candidate target genes of DLX3 over-expression and inactivation were identified as regulators of trophoblast differentiation; GATA2 and PPARγ. The expression of GATA2 and PPARγ were further assessed in placental tissues and showed increased mRNA and protein levels in FGR-affected tissues compared with gestation-matched controls. We conclude that DLX3 orchestrates the expression of multiple regulators of trophoblast differentiation and that expression of these regulatory genes is abnormal in FGR.

Keywords: Fetal growth; Homeobox genes; Human placenta; Trophoblast.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Cell Line
  • Female
  • Fetal Growth Retardation / genetics*
  • GATA2 Transcription Factor / metabolism
  • Gene Expression Profiling*
  • Gene Expression Regulation*
  • Genetic Association Studies
  • Homeodomain Proteins / genetics
  • Homeodomain Proteins / metabolism*
  • Humans
  • PPAR gamma / metabolism
  • Placenta / metabolism*
  • Placenta / pathology
  • Plasmids / metabolism
  • Pregnancy
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • RNA, Small Interfering / metabolism
  • Reproducibility of Results
  • Signal Transduction / genetics*
  • Transcription Factors / genetics
  • Transcription Factors / metabolism*
  • Trophoblasts / metabolism
  • Trophoblasts / pathology

Substances

  • Distal-less homeobox proteins
  • GATA2 Transcription Factor
  • GATA2 protein, human
  • Homeodomain Proteins
  • PPAR gamma
  • RNA, Messenger
  • RNA, Small Interfering
  • Transcription Factors