Role of cell cholesterol in modulating antineoplastic ether lipid uptake, membrane effects and cytotoxicity

Int J Cancer. 1990 Aug 15;46(2):341-6. doi: 10.1002/ijc.2910460234.

Abstract

Membrane-interactive ether lipids (EL) exert toxic and antiproliferative effects on cancer cells in vitro. They appear to be selectively more toxic to cancer cells than to normal cells and thus they are ideal candidates for bone-marrow purging procedures. However, no conclusive explanation has yet been provided for this property. We now present some data indicating that the cholesterol concentration in the incubation medium modulates EL toxicity against the HL60 leukemic cell line in vitro. Furthermore, model membranes richer in cholesterol take up EL more slowly, and cell cholesterol enrichment of HL60 cells counteracts EL biophysical membrane interaction, but not toxicity, in our experimental model. However, the K562 cell line, a leukemia line less sensitive to EL toxic action, has higher levels of cell cholesterol. Our data provide evidence to explain differences in sensitivity to EL among different cell types and contribute to the understanding of the mechanism of action of EL.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antineoplastic Agents / pharmacokinetics*
  • Antineoplastic Agents / toxicity
  • Cell Line
  • Cell Membrane / drug effects
  • Cell Membrane / metabolism*
  • Cholesterol / metabolism*
  • Dose-Response Relationship, Drug
  • Drug Interactions
  • Glyceryl Ethers / pharmacokinetics*
  • Glyceryl Ethers / toxicity
  • Humans
  • Leukemia / metabolism
  • Membrane Fluidity / drug effects
  • Phosphatidic Acids / pharmacokinetics*
  • Phosphatidic Acids / toxicity
  • Time Factors
  • Tumor Cells, Cultured / drug effects
  • Tumor Cells, Cultured / metabolism

Substances

  • Antineoplastic Agents
  • Glyceryl Ethers
  • Phosphatidic Acids
  • Cholesterol