[Association between MAOA-u VNTR polymorphism and its interaction with stressful life events and major depressive disorder in adolescents]

Zhongguo Dang Dai Er Ke Za Zhi. 2013 Jul;15(7):563-8.
[Article in Chinese]

Abstract

Objective: To investigate whether the genetic polymorphism, upstream variable number of tandem repeats (uVNTR), in the monoamine oxidase A (MAOA) gene, is associated with major depressive disorder (MDD) in adolescents and to test whether there is gene-environment interaction between MAOA-uVNTR polymorphism and stressful life events (SLEs).

Methods: A total of 394 Chinese Han subjects, including 187 adolescent patients with MDD and 207 normal students as a control group, were included in the study. Genotyping was performed by SNaP-shot assay. SLEs in the previous 12 months were evaluated. The groups were compared in terms of the frequency distributions of MAOA-uVNTR genotypes and alleles using statistical software. The binary logistic regression model of gene-environment interaction was established to analyze the association of the gene-environment interaction between MAOA-u VNTR genotypes and SLEs with adolescent MDD.

Results: The distribution profiles of MAOA-u VNTR genotypes and alleles were not related to the onset of MDD, severity of depression, comorbid anxiety and suicidal ideation/behavior/attempt in adolescents. The gene-environment interaction between MAOA-u VNTR genotypes and SLEs was not associated with MDD in male or female adolescents.

Conclusions: It is not proven that MAOA-u VNTR polymorphism is associated with adolescent MDD. There is also no gene-environment interaction between MAOA-u VNTR polymorphism and SLEs that is associated with adolescent MDD.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Depressive Disorder, Major / genetics*
  • Female
  • Gene-Environment Interaction*
  • Genotype
  • Humans
  • Life Change Events*
  • Logistic Models
  • Male
  • Minisatellite Repeats*
  • Monoamine Oxidase / genetics*
  • Polymorphism, Genetic*

Substances

  • Monoamine Oxidase