Selenium compounds induced ROS-dependent apoptosis in myelodysplasia cells

Biol Trace Elem Res. 2013 Sep;154(3):440-7. doi: 10.1007/s12011-013-9749-x. Epub 2013 Jul 31.

Abstract

Several authors have demonstrated the chemoprotective and anti-carcinogenic role of selenium. However, the therapeutic potential of selenium in myelodysplastic syndrome (MDS) as single agent and as co-adjuvant of the current therapies has not been previously studied. Sodium selenite and selenomethionine, alone and in combination with cytarabine, induce a decrease in cell viability in a time-, dose- and administration-dependent manner inducing cell death by apoptosis in F36P cells (MDS cell line). These compounds increased superoxide production and induced mitochondrial membrane depolarization. The increase in BAX/BCL-2 ratio and in the activated caspase 3 expression levels, the decrease in mitochondria membrane potential, as well as the increase in superoxide production, supports the mitochondria contribution on selenium-induced apoptosis. These findings suggest that selenium may offer a new therapeutic approach in myelodysplastic syndrome in monotherapy and/or as co-adjuvant therapy to conventional anti-carcinogenic.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antimetabolites, Antineoplastic / pharmacology
  • Apoptosis / drug effects*
  • Caspase 3 / metabolism
  • Cell Line
  • Cell Proliferation / drug effects*
  • Cell Survival / drug effects
  • Cytarabine / pharmacology
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Flow Cytometry
  • Humans
  • Membrane Potential, Mitochondrial / drug effects
  • Membrane Potential, Mitochondrial / physiology
  • Mitochondria / drug effects
  • Mitochondria / metabolism
  • Mitochondria / physiology
  • Myelodysplastic Syndromes / drug therapy
  • Myelodysplastic Syndromes / metabolism
  • Myelodysplastic Syndromes / pathology
  • Proto-Oncogene Proteins c-bcl-2 / metabolism
  • Reactive Oxygen Species / metabolism*
  • Selenium Compounds / pharmacology*
  • Selenomethionine / pharmacology
  • Sodium Selenite / pharmacology
  • Superoxides / metabolism
  • bcl-2-Associated X Protein / metabolism

Substances

  • Antimetabolites, Antineoplastic
  • BAX protein, human
  • Proto-Oncogene Proteins c-bcl-2
  • Reactive Oxygen Species
  • Selenium Compounds
  • bcl-2-Associated X Protein
  • Cytarabine
  • Superoxides
  • Selenomethionine
  • Caspase 3
  • Sodium Selenite