TAK-242 can be the potential agents for preventing invasion and metastasis of hepatocellular carcinoma

Med Hypotheses. 2013 Oct;81(4):653-5. doi: 10.1016/j.mehy.2013.06.034. Epub 2013 Jul 30.

Abstract

The long-term prognosis of hepatocellular carcinoma (HCC) remains unsatisfactory even after surgical resection and chemoembolization because of a high recurrence rate. However, fewer agents are currently available to block or postpone HCC progression. Recent studies show that the activation of toll-like receptor 4 (TLR4)-NF-kappaB(NF-κB) signaling pathway can lead to the expression of interleukin (IL)-1, IL-6, IL-10, nitric oxide (NO) and tumor necrosis factor (TNF)-α, and promote chronic liver inflammation and malignant tumor development. A new small molecule TAK-242 (Resatorevid) can selectively inhibit TLR4-mediated signaling pathway and suppress the expression of inflammatory mediators. But the researches have not been reported whether TAK-242 can inhibit liver tumor growth and invasion. So it suggested a new hypothesis that TAK-242 selectively blocks the TLR4-NF-κB pathway and is hoped to be a potential and creative drug for preventing invasion and metastasis of HCC.

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Carcinoma, Hepatocellular / drug therapy*
  • Carcinoma, Hepatocellular / physiopathology
  • Humans
  • Liver Neoplasms / drug therapy*
  • Liver Neoplasms / physiopathology
  • Models, Biological
  • NF-kappa B / metabolism
  • Neoplasm Invasiveness / prevention & control*
  • Neoplasm Metastasis / prevention & control*
  • Signal Transduction / drug effects
  • Sulfonamides / pharmacology*
  • Toll-Like Receptor 4 / metabolism

Substances

  • Antineoplastic Agents
  • NF-kappa B
  • Sulfonamides
  • TLR4 protein, human
  • Toll-Like Receptor 4
  • ethyl 6-(N-(2-chloro-4-fluorophenyl)sulfamoyl)cyclohex-1-ene-1-carboxylate