CX3CL1 protects neurons against excitotoxicity enhancing GLT-1 activity on astrocytes

J Neuroimmunol. 2013 Oct 15;263(1-2):75-82. doi: 10.1016/j.jneuroim.2013.07.020. Epub 2013 Aug 8.

Abstract

In this paper we show for the first time that: i) astrocytes are required for the neuroprotective activity of CX3CL1 against excitotoxicity; ii) inhibition of the glutamate transporter 1 (GLT-1) prejudices CX3CL1-mediated neuroprotection; iii) CX3CL1 increases GLT-1 activity on astrocytes. The modulation of GLT-1 activity induced by CX3CL1 on astrocytes requires the presence and the activity of A1 adenosine receptor (A1R), being blocked by the specific antagonist DPCPX and absent in A1R(-/-) astrocytes. These data introduce the astrocytes as active players in CX3CL1-mediated signaling between microglia and neurons, identifying GLT-1 as a key mediator of the neuroprotective activity of CX3CL1.

Keywords: A1R; Astrocytes; CX3CL1; GLT-1; Microglia; Neuroprotection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Newborn
  • Cell Survival / immunology
  • Cells, Cultured
  • Chemokine CX3CL1 / physiology*
  • Coculture Techniques
  • Excitatory Amino Acid Transporter 2 / antagonists & inhibitors*
  • Excitatory Amino Acid Transporter 2 / toxicity
  • Glucose / toxicity
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Neurons / cytology
  • Neurons / immunology*
  • Neurons / metabolism*
  • Neuroprotective Agents / therapeutic use*
  • Rats
  • Rats, Wistar
  • Up-Regulation / immunology*

Substances

  • Chemokine CX3CL1
  • Cx3cl1 protein, rat
  • Excitatory Amino Acid Transporter 2
  • Neuroprotective Agents
  • Slc1a2 protein, rat
  • Glucose