Biology of the estrogen receptor, GPR30, in triple negative breast cancer

Am J Surg. 2013 Nov;206(5):698-703. doi: 10.1016/j.amjsurg.2013.07.014. Epub 2013 Sep 4.

Abstract

Background: Triple-negative (TN) breast cancer lacks a known signaling pathway amenable to targeted therapy. The authors hypothesized that the G protein-coupled receptor GPR30 may be present in TN breast cancer and serve a role for tumor growth.

Methods: A retrospective pathology study and chart review were conducted. All patients aged ≤49 years from 2000 to 2008 were included (n = 24). Concurrent patients aged ≥50 years were randomly selected. Paraffin sections were stained for GPR30 and reviewed by a pathologist blinded to estrogen receptor and progesterone receptor status. Disease-free survival was analyzed versus age and receptor status. Means were compared using 2-sample t tests and proportions using chi-square analysis.

Results: Twenty-seven patients tested GPR30 positive and 21 GPR30 negative. Seventeen of 18 TN cancers tested positive for GPR30 (P < .0001). Recurrence at a mean follow-up of 36 months was 22.2% in the GPR30-positive group and 9.5% in the GPR30-negative group.

Conclusions: GPR30 is prevalent in TN breast cancer and associated with young age and possibly recurrence.

Keywords: Estrogen receptor; G protein–coupled receptor; Prognosis; Triple-negative breast cancer.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Factors
  • Female
  • Follow-Up Studies
  • Humans
  • Immunohistochemistry
  • Middle Aged
  • Neoplasm Recurrence, Local
  • Neoplasm Staging
  • Receptors, Estrogen / metabolism*
  • Receptors, G-Protein-Coupled / metabolism*
  • Retrospective Studies
  • Triple Negative Breast Neoplasms / metabolism*
  • Triple Negative Breast Neoplasms / pathology

Substances

  • GPER1 protein, human
  • Receptors, Estrogen
  • Receptors, G-Protein-Coupled