Ontogeny of excitatory spinal neurons processing distinct somatic sensory modalities

J Neurosci. 2013 Sep 11;33(37):14738-48. doi: 10.1523/JNEUROSCI.5512-12.2013.

Abstract

Spatial and temporal cues govern the genesis of a diverse array of neurons located in the dorsal spinal cord, including dI1-dI6, dIL(A), and dIL(B) subtypes, but their physiological functions are poorly understood. Here we generated a new line of conditional knock-out (CKO) mice, in which the homeobox gene Tlx3 was removed in dI5 and dIL(B) cells. In these CKO mice, development of a subset of excitatory neurons located in laminae I and II was impaired, including itch-related GRPR-expressing neurons, PKCγ-expressing neurons, and neurons expressing three neuropeptide genes: somatostatin, preprotachykinin 1, and the gastrin-releasing peptide. These CKO mice displayed marked deficits in generating nocifensive motor behaviors evoked by a range of pain-related or itch-related stimuli. The mutants also failed to exhibit escape response evoked by dynamic mechanical stimuli but retained the ability to sense innocuous cooling and/or warm. Thus, our studies provide new insight into the ontogeny of spinal neurons processing distinct sensory modalities.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Animals, Newborn
  • Capsaicin / toxicity
  • Cell Count
  • Chloroquine / toxicity
  • Embryo, Mammalian
  • Ganglia, Spinal / cytology*
  • Gastrin-Releasing Peptide / genetics
  • Gastrin-Releasing Peptide / metabolism
  • Gene Expression Regulation, Developmental / genetics
  • Gene Expression Regulation, Developmental / physiology*
  • Homeodomain Proteins / genetics
  • Mice
  • Mice, Transgenic
  • Muscle Proteins / genetics
  • Neurons / classification*
  • Neurons / physiology*
  • Oligopeptides / toxicity
  • Pain / chemically induced
  • Pain / genetics
  • Pain / metabolism
  • Physical Stimulation / adverse effects
  • Protein Kinase C / metabolism
  • Protein Precursors / genetics
  • Protein Precursors / metabolism
  • Pruritus / etiology
  • Pruritus / metabolism
  • Psychomotor Performance / drug effects
  • Psychomotor Performance / physiology
  • Sensation / genetics*
  • Somatostatin / genetics
  • Somatostatin / metabolism
  • Tachykinins / genetics
  • Tachykinins / metabolism
  • Vesicular Glutamate Transport Protein 1 / metabolism

Substances

  • Homeodomain Proteins
  • Lbx1h protein, mouse
  • Muscle Proteins
  • Oligopeptides
  • Protein Precursors
  • Slc17a7 protein, mouse
  • Tachykinins
  • Tlx3 protein, mouse
  • Vesicular Glutamate Transport Protein 1
  • preprotachykinin
  • seryl-leucyl-isoleucyl-glycyl--arginyl-leucinamide
  • Somatostatin
  • Gastrin-Releasing Peptide
  • Chloroquine
  • protein kinase C gamma
  • Protein Kinase C
  • Capsaicin