Neuronal development: SAD kinases make happy axons

Curr Biol. 2013 Sep 9;23(17):R720-3. doi: 10.1016/j.cub.2013.07.073.

Abstract

The polarity proteins LKB1 and SAD-A/B are key regulators of axon specification in the developing cerebral cortex. Recent studies now show that this mechanism cannot be generalized to other classes of neurons: instead, SAD-A/B functions downstream of neurotrophin signaling in sensory neurons to mediate a later stage of axon development - arborization in the target field.

Publication types

  • Comment

MeSH terms

  • Animals
  • Axons / metabolism*
  • Humans
  • Neurotrophin 3 / metabolism*
  • Protein Serine-Threonine Kinases / metabolism*
  • Sensory Receptor Cells / metabolism*
  • Signal Transduction / physiology*

Substances

  • Neurotrophin 3
  • Protein Serine-Threonine Kinases