A novel frameshift mutation in TWIST2 gene causing Setleis syndrome

Indian J Pediatr. 2014 Mar;81(3):302-4. doi: 10.1007/s12098-013-1253-y. Epub 2013 Oct 15.

Abstract

The authors report on a child with Setleis syndrome (OMIM 227260). She is born to a consanguineous couple with bitemporal scar like defects resembling forceps marks. She had other classical features resembling autosomal recessive Setleis syndrome. The authors identified a novel homozygous deletion of a single nucleotide (c.91delC) in TWIST2 gene leading to the premature truncation of protein (p.R31GfsX71). Umbilical hernia and genital anomalies are being reported for the first time with this condition. This is the fourth mutation proven family of Setleis syndrome.

Publication types

  • Case Reports
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Ectodermal Dysplasia
  • Female
  • Focal Dermal Hypoplasia / genetics*
  • Focal Facial Dermal Dysplasias
  • Frameshift Mutation / genetics*
  • Humans
  • Infant
  • Repressor Proteins / genetics*
  • Skin Diseases / genetics*
  • Twist-Related Protein 1 / genetics*

Substances

  • Repressor Proteins
  • TWIST2 protein, human
  • Twist-Related Protein 1