MCP-1 promotes invasion and adhesion of human ovarian cancer cells

Anticancer Res. 2013 Nov;33(11):4785-90.

Abstract

Background: Monocyte chemoattractant protein-1 (MCP-1) can accelerate tumor progression by attracting tumor-associated macrophages. We studied the effects of MCP-1 on SKOV-3 cells in order to investigate MCP-1 biological activity ovarian cancer.

Materials and methods: A SKOV-3 cell invasion assay (Transwell assay) and cell adhesion assay (96-well assay) were performed. Immunohistochemical staining for C-C motif chemokine receptor-2 (CCR2), a receptor for MCP-1, was also performed on cultured SKOV-3 cells.

Results: Migration and adhesion of MCP-1-treated SKOV-3 cells were significantly increased compared to untreated cells (p<0.01). A CCR2 antagonist attenuated the invasion and adhesion of MCP-1-treated cells. CCR2 was expressed in the cytoplasm of SKOV-3 cells.

Conclusion: MCP-1 promoted invasion and adhesion of ovarian cancer cells, and a CCR2 antagonist attenuated the effects of MCP-1 in vitro. These data suggest that MCP-1 is a potential therapeutic target for ovarian cancer therapy.

Keywords: C-C motif chemokine receptor-2 (CCR2) antagonist; Ovarian cancer; SKOV-3 cell line; monocyte chemoattractant protein-1 (MCP-1); tumor-associated macrophages (TAMs).

MeSH terms

  • Cell Adhesion*
  • Cell Movement*
  • Chemokine CCL2 / metabolism*
  • Cystadenocarcinoma, Serous / metabolism
  • Cystadenocarcinoma, Serous / pathology*
  • Female
  • Humans
  • Neoplasm Invasiveness
  • Ovarian Neoplasms / metabolism
  • Ovarian Neoplasms / pathology*
  • Receptors, CCR2 / antagonists & inhibitors*
  • Receptors, CCR2 / metabolism
  • Tumor Cells, Cultured

Substances

  • Chemokine CCL2
  • Receptors, CCR2