Requirement for recognition of class II molecules and processed tumor antigen for optimal generation of syngeneic tumor-specific class I-restricted CTL

J Immunol. 1986 Jun 1;136(11):4303-10.

Abstract

The roles of Class II-restricted L3T4+ T cells and of accessory cells (AC) during the in vitro generation of Class I-restricted Lyt-2+ cytotoxic T cells (CTL) specific for a Class II-negative syngeneic tumor cell line, FBL, was examined. Treatment of responder FBL-immune spleen cells with alpha L3T4 plus complement before culture, as well as the direct addition of alpha L3T4 to cultures, diminished the generation of FBL-specific CTL. The contribution of L3T4+ cells could be completely replaced by the addition of exogenous cytokines. The data demonstrate that the optimal generation of FBL-specific Lyt-2+ CTL requires the presence of L3T4+ cells, presumably to provide necessary lymphokines. FBL-specific CTL could not be generated from purified FBL-immune T cells in the absence of AC. Syngeneic Ia+ macrophages (M phi), added at the initiation of culture, restored the response of purified T cells. Pretreatment of M phi with ammonium chloride or chloroquine, or the addition of monoclonal alpha I-Ab antibody at the initiation of culture, inhibited the ability of M phi to reconstitute the CTL response. Finally, the addition of exogenous helper factors could replace M phi and reconstitute the FBL-specific response of AC-depleted immune T cells. These results suggest that during the generation of Lyt-2+ CTL to a syngeneic tumor expressing only Class I MHC antigens, Ia+ AC are required to biochemically process antigen released from the tumor cells and present this modified antigen to Class II-restricted T helper cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigen-Presenting Cells / immunology
  • Antigen-Presenting Cells / metabolism*
  • Antigens, Differentiation, T-Lymphocyte
  • Antigens, Ly / analysis
  • Antigens, Neoplasm / analysis*
  • Antigens, Neoplasm / immunology
  • Antigens, Surface / analysis
  • Cell Communication
  • Epitopes / immunology
  • Female
  • H-2 Antigens / analysis
  • H-2 Antigens / genetics*
  • Histocompatibility Antigens Class II / analysis*
  • Histocompatibility Antigens Class II / immunology
  • Leukemia, Erythroblastic, Acute / immunology
  • Lymphocyte Activation
  • Mice
  • Mice, Inbred AKR
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Mice, Inbred CBA
  • Mice, Nude
  • Monokines
  • Proteins / pharmacology
  • T-Lymphocytes, Cytotoxic / immunology*
  • T-Lymphocytes, Cytotoxic / metabolism

Substances

  • Antigens, Differentiation, T-Lymphocyte
  • Antigens, Ly
  • Antigens, Neoplasm
  • Antigens, Surface
  • Epitopes
  • H-2 Antigens
  • Histocompatibility Antigens Class II
  • Monokines
  • Proteins