Motor protein function in skeletal abdominal muscle of cachectic cancer patients

J Cell Mol Med. 2014 Jan;18(1):69-79. doi: 10.1111/jcmm.12165. Epub 2013 Nov 19.

Abstract

Cachexia presents with ongoing muscle wasting, altering quality of life in cancer patients. Cachexia is a limiting prognostic factor for patient survival and health care costs. Although animal models and human trials have shown mechanisms of motorprotein proteolysis, not much is known about intrinsic changes of muscle functionality in cancer patients suffering from muscle cachexia, and deeper insights into cachexia pathology in humans are needed. To address this question, rectus abdominis muscle samples were collected from several surgical control, non-cachectic and cachectic cancer patients and processed for skinned fibre biomechanics, molecular in vitro motility assays, myosin isoform protein compositions and quantitative ubiquitin polymer protein analysis. In pre-cachectic and cachectic cancer patient samples, maximum force was significantly compromised compared with controls, but showed an unexpected increase in myofibrillar Ca(2+) sensitivity consistent with a shift from slow to fast myosin isoform expression seen in SDS-PAGE analysis and in vitro motility assays. Force deficit was specific for 'cancer', but not linked to presence of cachexia. Interestingly, quantitative ubiquitin immunoassays revealed no major changes in static ubiquitin polymer protein profiles, whether cachexia was present or not and were shown to mirror profiles in control patients. Our study on muscle function in cachectic patients shows that abdominal wall skeletal muscle in cancer cachexia shows signs of weakness that can be partially attributed to intrinsic changes to contractile motorprotein function. On protein levels, static ubiquitin polymeric distributions were unaltered, pointing towards evenly up-regulated ubiquitin protein turnover with respect to ubiquitin conjugation, proteasome degradation and de-ubiquitination.

Keywords: cancer; contractility; motility assay; muscle cachexia; myosin isoforms.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Abdominal Muscles / metabolism*
  • Abdominal Muscles / pathology
  • Abdominal Muscles / physiopathology
  • Adult
  • Aged
  • Aged, 80 and over
  • Cachexia / etiology
  • Cachexia / metabolism*
  • Cachexia / physiopathology
  • Calcium / physiology
  • Colonic Neoplasms / complications
  • Colonic Neoplasms / metabolism*
  • Colonic Neoplasms / physiopathology
  • Female
  • Humans
  • In Vitro Techniques
  • Male
  • Middle Aged
  • Muscle Contraction
  • Myosins / physiology*
  • Pancreatic Neoplasms / complications
  • Pancreatic Neoplasms / metabolism*
  • Pancreatic Neoplasms / physiopathology
  • Protein Isoforms / metabolism
  • Single-Cell Analysis
  • Ubiquitin / metabolism

Substances

  • Protein Isoforms
  • Ubiquitin
  • Myosins
  • Calcium