Renal function can improve at any stage of chronic kidney disease

PLoS One. 2013 Dec 13;8(12):e81835. doi: 10.1371/journal.pone.0081835. eCollection 2013.

Abstract

Introduction: Even though renal function decline is considered relentless in chronic kidney disease (CKD), improvement has been shown in patients with hypertensive nephropathy. Whether this can occur in any type of nephropathy and at any stage is unknown as are the features of patients who improve.

Methods: We identified 406 patients in the NephroTest cohort with glomerular filtration rates (mGFR) measured by (51)Cr-EDTA clearance at least 3 times during at least 2 years of follow-up. Individual examination of mGFR trajectories by 4 independent nephrologists classified patients as improvers, defined as those showing a sustained mGFR increase, or nonimprovers. Twelve patients with erratic trajectories were excluded. Baseline data were compared between improvers and nonimprovers, as was the number of recommended therapeutic targets achieved over time (specifically, for systolic and diastolic blood pressure, proteinuria, and use of renin angiotensin system blockers).

Results: Measured GFR improved over time in 62 patients (15.3%). Their median mGFR slope was +1.88[IQR 1.38, 3.55] ml/min/year; it was -2.23[-3.9, -0.91] for the 332 nonimprovers. Improvers had various nephropathies, but not diabetic glomerulopathy or polycystic kidney disease. They did not differ from nonimprovers for age, sex, cardiovascular history, or CKD stage, but their urinary albumin excretion rate was lower. Improvers achieved significantly more recommended therapeutic targets (2.74±0.87) than nonimprovers (2.44±0.80, p<0.01). They also had fewer CKD-related metabolic complications and a lower prevalence of 25OH-vitamin-D deficiency.

Conclusion: GFR improvement is possible in CKD patients at any CKD stage through stage 4-5. It is noteworthy that this GFR improvement is associated with a decrease in the number of metabolic complications over time.

MeSH terms

  • Adult
  • Aged
  • Albuminuria / complications
  • Albuminuria / diagnosis
  • Albuminuria / drug therapy
  • Albuminuria / physiopathology*
  • Angiotensin-Converting Enzyme Inhibitors / therapeutic use
  • Blood Pressure
  • Chronic Disease
  • Disease Progression
  • Female
  • Glomerular Filtration Rate / physiology*
  • Humans
  • Hypertension, Renal / complications
  • Hypertension, Renal / diagnosis
  • Hypertension, Renal / drug therapy
  • Hypertension, Renal / physiopathology*
  • Kidney
  • Male
  • Middle Aged
  • Nephritis / complications
  • Nephritis / diagnosis
  • Nephritis / drug therapy
  • Nephritis / physiopathology*
  • Renal Insufficiency, Chronic / complications
  • Renal Insufficiency, Chronic / diagnosis
  • Renal Insufficiency, Chronic / drug therapy
  • Renal Insufficiency, Chronic / physiopathology*
  • Renin-Angiotensin System / drug effects
  • Severity of Illness Index
  • Treatment Outcome
  • Vitamin D Deficiency / complications
  • Vitamin D Deficiency / diagnosis
  • Vitamin D Deficiency / drug therapy
  • Vitamin D Deficiency / physiopathology*

Substances

  • Angiotensin-Converting Enzyme Inhibitors

Supplementary concepts

  • Hypertensive Nephropathy

Grants and funding

The NephroTest CKD cohort study is supported by grants from: Inserm GIS-IReSP AO 8113LS TGIR (BS); French Ministry of Health AOM 09114 (MF); Inserm AO 8022LS (BS); Agence de la Biomédecine R0 8156LL (BS); AURA (MFr) and Roche 2009-152-447G (MFr). The Nephrotest initiative was also sponsored by unrestricted grants from F. Hoffman-La Roche Ltd. (LM). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.