Identification and sequencing of cDNA clones for the rodent negative acute-phase protein alpha 1-inhibitor 3

Eur J Biochem. 1987 Apr 15;164(2):375-81. doi: 10.1111/j.1432-1033.1987.tb11068.x.

Abstract

Rat alpha 1-inhibitor 3 clones were isolated by immunological screening of a lambda gt11 cDNA library prepared from rat liver poly(A)-rich RNA. The recombinant cDNA clones were identified by the absence of their immunoprecipitable products following hybrid-arrested in vitro translation. The size of the cognate poly(A)-rich RNA was estimated to be roughly 5000 residues. Approximately 16 h after induction of inflammation the amount of alpha 1-inhibitor 3 poly(A)-rich RNA decreases as shown by dot-blot hybridization and Northern analyses. The response of this negative acute-phase plasma protein to inflammation may therefore be considered to be at the pretranslational level. The characterized DNA constitutes an open reading frame of 225 amino acids followed by a canonical eucaryotic polyadenylation signal and a poly(A) tail. Sequence microheterogeneity, particularly in the 3'-flanking region was observed. An amino acid homology of 70% for alpha 1-inhibitor 3 with human and rodent alpha 2-macroglobulin emphasizes the evolutionary relationship of the macroglobulins.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute-Phase Proteins*
  • Amino Acid Sequence
  • Animals
  • Base Sequence
  • Biological Evolution
  • Cloning, Molecular
  • DNA / genetics*
  • Humans
  • Protease Inhibitors / genetics*
  • Rats
  • alpha-Macroglobulins / genetics

Substances

  • Acute-Phase Proteins
  • Protease Inhibitors
  • alpha 1-inhibitor 3
  • alpha-Macroglobulins
  • DNA