Downregulation of CD4+LAP+ and CD4+CD25+ regulatory T cells in acute coronary syndromes

Mediators Inflamm. 2013:2013:764082. doi: 10.1155/2013/764082. Epub 2013 Dec 10.

Abstract

Background: Regulatory T (Treg) cells play a protective role in atherosclerosis prone models and are related to the onset of acute coronary syndromes (ACS, including non-ST-elevation ACS (NSTEACS) and ST-elevation acute myocardial infarction (STEAMI)). CD4+LAP+ Treg cells are a novel subset of Tregs that have been found to ameliorate atherosclerosis in ApoE(-/-) mice, and these cells also exist in humans. The present study was designed to investigate whether CD4+LAP+ Treg cells are involved in the onset of ACS.

Methods: The frequencies of CD4+LAP+ and CD4+CD25+ Treg cells were detected using flow cytometric analysis, and the plasma IL-10 and TGF- β 1 levels were measured using an ELISA in 29 stable angina (SA) patients, 30 NSTEACS patients, 27 STEAMI patients, and a control group (30 cases).

Results: The results revealed a significant decrease in the frequencies of CD4+LAP+ and CD4+CD25+ Treg cells and in the levels of IL-10 and TGF- β 1 in patients with ACS compared with those in the SA and control groups.

Conclusions: The decrease in the frequencies of CD4+LAP+ and CD4+CD25+ Treg cells may play a role in the onset of ACS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Coronary Syndrome / immunology*
  • Acute Coronary Syndrome / physiopathology
  • Aged
  • Down-Regulation
  • Female
  • Forkhead Transcription Factors / physiology
  • Humans
  • Interleukin-10 / blood
  • Male
  • Middle Aged
  • Peptides / physiology*
  • Protein Precursors / physiology*
  • T-Lymphocytes, Regulatory / immunology*
  • Transforming Growth Factor beta / physiology*
  • Transforming Growth Factor beta1 / blood
  • Ventricular Function, Left

Substances

  • FOXP3 protein, human
  • Forkhead Transcription Factors
  • Peptides
  • Protein Precursors
  • Transforming Growth Factor beta
  • Transforming Growth Factor beta1
  • latency-associated propeptide, TGF-beta
  • Interleukin-10