Extracellular Ca2+ induces a rapid increase in cytoplasmic free Ca2+ in pancreatic beta-cells

Biochem Biophys Res Commun. 1987 Nov 30;149(1):152-8. doi: 10.1016/0006-291x(87)91617-2.

Abstract

Using pancreatic beta-cells isolated from obese hyperglycemic mice, it was demonstrated that the addition of 5 mM extracellular Ca2+ evoked a rapid and transient increase in cytoplasmic free Ca2+ concentration ([Ca2+]i). The effect remained in the presence of D-600. Extracellular Ca2+ did not raise [Ca2+]i subsequent to emptying the inositol 1,4,5-trisphosphate (InsP3) sensitive pool by carbamylcholine stimulation, indicating that the pool released by extracellular Ca2+ is of similar origin. Stimulation with extracellular Ca2+ was accompanied by a pronounced insulin release. Our results suggest that the Ca2+-induced rise in [Ca2+]i is mediated through the formation of InsP3, a mechanism that might operate also in other types of cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Barium / pharmacology
  • Calcium / physiology*
  • Carbachol / pharmacology
  • Cations, Divalent
  • Cytoplasm / metabolism
  • Extracellular Space / metabolism
  • Female
  • Gallopamil / pharmacology
  • Glucose / pharmacology
  • Insulin / metabolism
  • Insulin Secretion
  • Ion Channels / drug effects
  • Ion Channels / metabolism
  • Islets of Langerhans / drug effects
  • Islets of Langerhans / metabolism*
  • Magnesium / pharmacology
  • Male
  • Mice
  • Mice, Obese
  • Strontium / pharmacology

Substances

  • Cations, Divalent
  • Insulin
  • Ion Channels
  • Barium
  • Gallopamil
  • Carbachol
  • Magnesium
  • Glucose
  • Calcium
  • Strontium