Clinical and preclinical characterization of the histamine H(4) receptor antagonist JNJ-39758979

J Pharmacol Exp Ther. 2014 May;349(2):176-84. doi: 10.1124/jpet.113.211714. Epub 2014 Feb 18.

Abstract

The histamine H4 receptor (H(4)R) has been shown to have preclinical involvement in both inflammatory and pruritic responses. JNJ-39758979 [(R)-4-(3-amino-pyrrolidin-1-yl)-6-isopropyl-pyrimidin-2-ylamine] is a potent and selective H(4)R antagonist with a Ki at the human receptor of 12.5 ± 2.6 nM and greater than 80-fold selectivity over other histamine receptors. The compound also exhibited excellent selectivity versus other targets. JNJ-39758979 showed dose-dependent activity in models of asthma and dermatitis consistent with other H(4)R antagonists. Preclinical toxicity studies of up to 6 months in rats and 9 months in monkeys indicated an excellent safety profile, supporting the clinical testing of the compound. An oral formulation of JNJ-39758979 was studied in a phase 1 human volunteer study to assess safety, pharmacokinetics, and pharmacodynamics. The compound was well tolerated, with the exception of dose-dependent nausea, and no safety issues were noted in the phase 1 study. JNJ-39758979 exhibited good pharmacokinetics upon oral dosing with a plasma half-life of 124-157 hours after a single oral dose. In addition, dose-dependent inhibition of histamine-induced eosinophil shape change was detected, suggesting that the H4R was inhibited in vivo. In conclusion, JNJ-39758979 is a potent and selective H(4)R antagonist that exhibited good preclinical and phase 1 safety in healthy volunteers with evidence of a pharmacodynamics effect in humans.

Publication types

  • Clinical Trial, Phase I
  • Randomized Controlled Trial

MeSH terms

  • Animals
  • Anti-Inflammatory Agents / adverse effects
  • Anti-Inflammatory Agents / pharmacokinetics
  • Anti-Inflammatory Agents / pharmacology
  • Asthma / drug therapy
  • Asthma / immunology
  • Asthma / pathology
  • Cell Shape / drug effects
  • Dermatitis, Contact / drug therapy
  • Dermatitis, Contact / etiology
  • Dermatitis, Contact / immunology
  • Double-Blind Method
  • Eosinophils / drug effects
  • Eosinophils / pathology
  • Female
  • Humans
  • Isothiocyanates
  • Macaca fascicularis
  • Male
  • Ovalbumin
  • Pyrimidines / adverse effects
  • Pyrimidines / pharmacokinetics
  • Pyrimidines / pharmacology*
  • Rats
  • Receptors, G-Protein-Coupled / antagonists & inhibitors*
  • Receptors, Histamine
  • Receptors, Histamine H4
  • Thiazoles / adverse effects
  • Thiazoles / pharmacokinetics
  • Thiazoles / pharmacology*

Substances

  • (2-(2,6-dichlorobenzyl)thiazolo(5,4-d)pyrimidin-7-yl)-(4-trifluoromethylphenyl)amine
  • Anti-Inflammatory Agents
  • HRH4 protein, human
  • Isothiocyanates
  • Pyrimidines
  • Receptors, G-Protein-Coupled
  • Receptors, Histamine
  • Receptors, Histamine H4
  • Thiazoles
  • isothiocyanic acid
  • Ovalbumin