Evidence for decreased expression of ADAMTS-1 associated with impaired oocyte quality in PCOS patients

J Clin Endocrinol Metab. 2014 Jun;99(6):E1015-21. doi: 10.1210/jc.2013-4177. Epub 2014 Mar 19.

Abstract

Context: Polycystic ovary syndrome (PCOS) is the most common cause of dysfunctional ovulation-affecting female fertility. A disintegrin and metalloproteinase with thrombospondin-like motifs (ADAMTS-1) is required for normal ovulation and subsequent fertilization, and the expression of ADAMTS-1 may be altered in PCOS granulosa cell (GC)-reflecting abnormalities in ovulatory signaling.

Objective: The purpose of this paper is to analyze the differential expression of ADAMTS-1 in PCOS patients associated with impaired oocyte quality.

Design and setting: A prospective comparative experimental study was performed at a clinical reproductive medicine center.

Patients: Women with PCOS (n = 40) and normovulatory controls (n = 40) undergoing controlled ovarian hyperstimulation and in vitro fertilization were recruited in our study.

Main outcome measures: Differential expression of ADAMTS-1 in GCs was analyzed with immunocytochemistry in PCOS patients and normal controls, and ADAMTS-1 mRNA expression was quantified by RT-PCR. Furthermore, the correlation between ADAMTS-1 mRNA and oocyte quality was analyzed.

Results: The expression of ADAMTS-1 was decreased in PCOS patients compared with normally ovulating women and was closely related to lower oocyte recovery, oocyte maturity, and fertilization rate.

Conclusion: Our study provides evidence that the dysregulated expression of ADAMTS-1 in PCOS may influence oocyte quality-via GCs-oocyte paracrine and endocrine mechanism.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • ADAM Proteins / genetics*
  • ADAM Proteins / metabolism
  • ADAMTS1 Protein
  • Adult
  • Cells, Cultured
  • Down-Regulation
  • Female
  • Fertilization in Vitro
  • Granulosa Cells / metabolism
  • Hep G2 Cells
  • Humans
  • Oocyte Retrieval
  • Oocytes / metabolism
  • Oocytes / pathology*
  • Ovulation Induction
  • Polycystic Ovary Syndrome / genetics*
  • Polycystic Ovary Syndrome / pathology*
  • Pregnancy
  • Quality Control

Substances

  • ADAM Proteins
  • ADAMTS1 Protein
  • ADAMTS1 protein, human