The physiopathological role of IL-33: new highlights in bone biology and a proposed role in periodontal disease

Mediators Inflamm. 2014:2014:342410. doi: 10.1155/2014/342410. Epub 2014 Feb 18.

Abstract

Interleukin-33 (IL-33) is a recently described member of the IL-1 family. IL-33 acts as an alarmin, chemoattractant, and nuclear factor. ST2, a member of the Toll-like receptor/IL-1R superfamily, the receptor of IL-33, triggers a plethora of downstream effectors and leads the activation of NFK-B, leading the expression of several genes. IL-33 and ST2 are expressed in the majority of cell types, and the IL-33/ST2 axis has a role in immune response, bone homeostasis, and osteoclastogenesis. Several studies show opposite roles of IL-33 in osteoclastogenesis and the implication in bone biology. Few works studied the role of IL-33 in periodontal disease, but we hypothesize a possible role of IL-33 in periodontal disease and bone loss.

Publication types

  • Review

MeSH terms

  • Bone and Bones / metabolism*
  • Homeostasis
  • Humans
  • Immune System
  • Inflammation
  • Interleukin-1 / metabolism
  • Interleukin-1 Receptor-Like 1 Protein
  • Interleukin-33
  • Interleukins / physiology*
  • Mast Cells / cytology
  • Necrosis
  • Osteoclasts / cytology
  • Periodontal Diseases / metabolism*
  • Periodontal Diseases / physiopathology*
  • RANK Ligand / metabolism
  • Receptors, Cell Surface / metabolism
  • Signal Transduction

Substances

  • IL1RL1 protein, human
  • IL33 protein, human
  • Interleukin-1
  • Interleukin-1 Receptor-Like 1 Protein
  • Interleukin-33
  • Interleukins
  • RANK Ligand
  • Receptors, Cell Surface