Autoantibodies induced by chimeric cytokine-HIV envelope glycoprotein immunogens

J Immunol. 2014 May 15;192(10):4628-35. doi: 10.4049/jimmunol.1303401. Epub 2014 Apr 11.

Abstract

Cytokines are often used as adjuvants to increase the immunogenicity of vaccines because they can improve the immune response and/or direct it into a desired direction. As an alternative to codelivering Ags and cytokines separately, they can be fused into a composite protein, with the advantage that both moieties act on the same immune cells. The HIV-1 envelope glycoprotein (Env) spike, located on the outside of virus particles and the only relevant protein for the induction of neutralizing Abs, is poorly immunogenic. The induction of anti-Env Abs can be improved by coupling Env proteins to costimulatory molecules such as a proliferation inducing ligand (APRIL). In this study, we evaluated the immunogenicity of chimeric molecules containing uncleaved Env gp140 fused to the species-matched cytokines IL-21 or GM-CSF in rabbits and mice. Each cytokine was either fused to the C terminus of Env or embedded within Env at the position of the variable loops 1 and 2. The cytokine components of the chimeric Env-GM-CSF and Env-IL-21 molecules were functional in vitro, but none of the Env-cytokine fusion proteins resulted in improved Ab responses in vivo. Both the Env-GM-CSF and the Env-IL-21 molecules induced strong anticytokine Ab responses in both test species. These autoimmune responses were independent of the location of the cytokine in the chimeric Env molecules in that they were induced by cytokines inserted within the variable loops 1 and 2 of Env or fused to its C terminus. The induction of undesired autoimmune responses should be considered when using cytokines as costimulatory molecules in fusion proteins.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autoantibodies / immunology*
  • Autoimmune Diseases* / chemically induced
  • Autoimmune Diseases* / genetics
  • Autoimmune Diseases* / immunology
  • Autoimmune Diseases* / pathology
  • Cell Line, Tumor
  • HIV-1* / genetics
  • HIV-1* / immunology
  • Humans
  • Interleukins* / adverse effects
  • Interleukins* / genetics
  • Interleukins* / immunology
  • Interleukins* / pharmacology
  • Mice
  • Protein Structure, Secondary
  • Rabbits
  • Recombinant Fusion Proteins* / adverse effects
  • Recombinant Fusion Proteins* / genetics
  • Recombinant Fusion Proteins* / immunology
  • Recombinant Fusion Proteins* / pharmacology
  • env Gene Products, Human Immunodeficiency Virus* / adverse effects
  • env Gene Products, Human Immunodeficiency Virus* / genetics
  • env Gene Products, Human Immunodeficiency Virus* / immunology
  • env Gene Products, Human Immunodeficiency Virus* / pharmacology

Substances

  • Autoantibodies
  • Interleukins
  • Recombinant Fusion Proteins
  • env Gene Products, Human Immunodeficiency Virus
  • interleukin-21