ParB spreading requires DNA bridging

Genes Dev. 2014 Jun 1;28(11):1228-38. doi: 10.1101/gad.242206.114. Epub 2014 May 14.

Abstract

The parABS system is a widely employed mechanism for plasmid partitioning and chromosome segregation in bacteria. ParB binds to parS sites on plasmids and chromosomes and associates with broad regions of adjacent DNA, a phenomenon known as spreading. Although essential for ParB function, the mechanism of spreading remains poorly understood. Using single-molecule approaches, we discovered that Bacillus subtilis ParB (Spo0J) is able to trap DNA loops. Point mutants in Spo0J that disrupt DNA bridging are defective in spreading and recruitment of structural maintenance of chromosomes (SMC) condensin complexes in vivo. DNA bridging helps to explain how a limited number of Spo0J molecules per parS site (~20) can spread over many kilobases and suggests a mechanism by which ParB proteins could facilitate the loading of SMC complexes. We show that DNA bridging is a property of diverse ParB homologs, suggesting broad evolutionary conservation.

Keywords: ParB; Spo0J; bacterial chromosome segregation; single-molecule fluorescence.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Bacillus subtilis / genetics*
  • Bacillus subtilis / metabolism*
  • Bacterial Proteins / metabolism
  • Cell Cycle Proteins / metabolism
  • Cell Nucleus Shape / genetics
  • DNA Primase / genetics
  • DNA Primase / metabolism*
  • DNA, Bacterial / metabolism*
  • Point Mutation
  • Protein Binding

Substances

  • Bacterial Proteins
  • Cell Cycle Proteins
  • DNA, Bacterial
  • SMC protein, Bacteria
  • DNA Primase