Control of C4a-hydroperoxyflavin protonation in the oxygenase component of p-hydroxyphenylacetate-3-hydroxylase

Biochemistry. 2014 Jul 1;53(25):4084-6. doi: 10.1021/bi500480n. Epub 2014 Jun 17.

Abstract

The protonation status of the peroxide moiety in C4a-(hydro)peroxyflavin of p-hydroxyphenylacetate-3-hydroxylase can be directly monitored using transient kinetics. The pKa for the wild-type (WT) enzyme is 9.8 ± 0.2, while the values for the H396N, H396V, and H396A variants are 9.3 ± 0.1, 7.3 ± 0.2, and 7.1 ± 0.2, respectively. The hydroxylation efficiency of these mutants is lower than that of the WT enzyme. Solvent kinetic isotope effect studies indicate that proton transfer is not the rate-limiting step in the formation of C4a-OOH. All data suggest that His396 may act as an instantaneous proton provider for the proton-coupled electron transfer that occurs before the transition state of C4a-OOH formation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Flavins / chemistry*
  • Hydrogen-Ion Concentration
  • Kinetics
  • Mixed Function Oxygenases / chemistry*
  • Mixed Function Oxygenases / genetics
  • Mutation
  • Protons*

Substances

  • C4a-hydroperoxyflavin
  • Flavins
  • Protons
  • Mixed Function Oxygenases
  • 4-hydroxyphenylacetate 3-monooxygenase