Usefulness of decrease in oxygen uptake efficiency to identify gas exchange abnormality in patients with idiopathic pulmonary arterial hypertension

PLoS One. 2014 Jun 6;9(6):e98889. doi: 10.1371/journal.pone.0098889. eCollection 2014.

Abstract

Background: Decline in oxygen uptake efficiency (OUE), especially during exercise, is found in patients with chronic heart failure. In this study we aimed to test the validity and usefulness of OUE in evaluating gas exchange abnormality of patients with idiopathic pulmonary arterial hypertension (IPAH).

Methods: We retrospectively investigated the cardiopulmonary exercise test (CPET) with gas exchange measurements in 32 patients with confirmed IPAH. All patients also had resting hemodynamic measurements and pulmonary function test (PFT). Sixteen healthy subjects, matched by age, sex, and body size were used as controls, also had CPET and PFT measurements.

Results: In IPAH patients, the magnitude of absolute and percentage of predicted (%pred) oxygen uptake efficiency slope (OUES) and oxygen uptake efficiency plateau (OUEP), as well as several other CPET parameters, were strikingly worse than healthy subjects (P<0.0001). Pattern of changes in OUE in patients is similar to that in controls, In IPAH patients, OUE values at rest, warming up, anaerobic threshold and peak exercise were all significantly lower than in normal (P<0.0001). OUEP%pred, better than OUES%pred, correlated significantly with New York Heart Association (NYHA) functional Class (r = -0.724, P<0.005), Total Pulmonary Vascular Resistance (TPVR) (r = -0.694, P<0.005), diffusing capacity for carbon monoxide (DLCO) (r = 0.577, P<0.05), and the lowest ventilation versus CO2 output ratio during exercise (LowestV˙E/V˙CO2) (r = -0.902, P<0.0001). In addition, the coefficient of variation (COV) of OUEP was lower (20.9%) markedly than OUES (34.3%) (P<0.0001).

Conclusions: In patients with IPAH, OUES and OUEP are both significantly lower than the healthy subjects. OUEP is a better physiological parameter than OUES in evaluating the gas exchange abnormality of patients with IPAH.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Cardiac Output
  • Case-Control Studies
  • Exercise / physiology
  • Familial Primary Pulmonary Hypertension / metabolism*
  • Familial Primary Pulmonary Hypertension / physiopathology
  • Female
  • Humans
  • Male
  • Oxygen Consumption*
  • Pulmonary Gas Exchange*
  • Pulmonary Ventilation
  • Rest / physiology
  • Retrospective Studies

Grants and funding

This retrospective CPET data reanalysis is partially supported by Chinese Academy of Medical Sciences (No. 2012-YJR02), Key Project Starting Grant from National Center for Cardiovascular Diseases. CPET data collected at Shanghai Pulmonary Hospital were partially supported from Chinese Medicine Association (No. 08020420120) and science and technology commission of shanghai municipality (NO. 11411951302 and 114119a3000). The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.