Microsomal lipid peroxidation: mechanisms of initiation. The role of iron and iron chelators

Free Radic Biol Med. 1989;6(1):31-6. doi: 10.1016/0891-5849(89)90156-1.

Abstract

The role of iron and iron chelators in the initiation of microsomal lipid peroxidation has been investigated. It is shown that an Fe3+ chelate in order to be able to initiate enzymically induced lipid peroxidation in rat liver microsomes has to fulfill three criteria: (a) reducibility by NADPH; (b) reactivity of the Fe2+ chelate with rat liver microsomes has to fulfill three criteria: (a) reducibility by NADPH; (b) reactivity of the Fe2+ chelate with O2; and (c) formation of a relatively stable perferryl radical. NADH can support lipid peroxidation in the presence of ADP-Fe3+ or oxalate-Fe3+ at rates comparable to those obtained with NADPH but requires 10 to 15 times higher concentrations of the Fe3+ chelates for maximal activity. The results are discussed in relation to earlier proposed mechanisms of microsomal lipid peroxidation.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenosine Diphosphate / pharmacology
  • Adenosine Triphosphate / pharmacology
  • Animals
  • Edetic Acid / pharmacology
  • Free Radicals
  • Iron / metabolism
  • Iron / pharmacology*
  • Iron Chelating Agents / metabolism
  • Iron Chelating Agents / pharmacology*
  • Lipid Peroxidation / drug effects*
  • Malonates / pharmacology
  • Microsomes, Liver / metabolism*
  • NAD / metabolism
  • NADP / metabolism
  • Oxalates / pharmacology
  • Oxalic Acid
  • Oxidation-Reduction
  • Oxygen / metabolism
  • Phosphates / pharmacology
  • Rats

Substances

  • Free Radicals
  • Iron Chelating Agents
  • Malonates
  • Oxalates
  • Phosphates
  • NAD
  • NADP
  • Adenosine Diphosphate
  • Adenosine Triphosphate
  • Oxalic Acid
  • Edetic Acid
  • malonic acid
  • Iron
  • Oxygen