Phosphatidylinositol turnover is not a general regulator of neuroblastoma cell differentiation: comparison between two differentiating agents, retinoic acid and gamma-interferon

FEBS Lett. 1989 Jan 30;243(2):285-8. doi: 10.1016/0014-5793(89)80146-2.

Abstract

The turnover of phosphatidylinositol (PI) is believed to constitute a crucial step in the signaling pathways for stimulation of cells by a variety of bioactive substances, including differentiating agents; however decisive evidence for the idea has not been obtained. In the present paper, we investigated the involvement of PI turnover in cell differentiation using a human neuroblastoma cell line, LAN-1, which can be induced to differentiate along the neuronal pathway by both retinoic acid (RA) and gamma-interferon (gamma-IFN). Analysis of labelled phosphatidylinositol metabolites from prelabelled cells indicated a rapid decrease of inositol 1,4,5-trisphosphate and 1,2-diacylglycerol within 1 min of induction of LAN-1 cell differentiation by RA, while no changes were observed in gamma-IFN-treated cells. These findings indicate the occurrence of decreased inositol phospholipid turnover in RA-treated LAN-1 cells and suggest that phosphoinositide-derived metabolites may not constitute general regulators of cellular differentiation.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation / drug effects*
  • Chromatography, High Pressure Liquid / methods
  • Chromatography, Thin Layer
  • Humans
  • Interferon-gamma / pharmacology*
  • Neuroblastoma
  • Phosphatidylinositols / metabolism*
  • Phosphatidylinositols / physiology
  • Tretinoin / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Phosphatidylinositols
  • Tretinoin
  • Interferon-gamma