Activation of latent K+ uniport in mitochondria treated with the ionophore A23187

J Biol Chem. 1989 Nov 15;264(32):18902-6.

Abstract

Addition of A23187 plus EDTA to energized mitochondria in KCl medium determines a rapid osmotic swelling due to K+ uptake. The swelling is fully reversed by uncoupler, is stimulated by quinine, and is accompanied by membrane depolarization and increased rate of respiration. A23187-treated mitochondria passively swell in K+ thiocyanate at neutral pH, under conditions where the H+-K+ antiporter appears to be silent. These data indicate that A23187 activates electrophoretic K+ flux, supporting the notion that Mg2+ depletion unmasks several ionic conductance pathways whose concerted interplay could provide a sensitive regulation of mitochondrial volume homeostasis.

MeSH terms

  • Calcimycin / pharmacology*
  • Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone / pharmacology
  • Intracellular Membranes / drug effects
  • Intracellular Membranes / physiology
  • Kinetics
  • Membrane Potentials / drug effects
  • Mitochondria / drug effects
  • Mitochondria / metabolism*
  • Mitochondrial Swelling / drug effects
  • Potassium / metabolism*
  • Potassium / pharmacology
  • Potassium Chloride / pharmacology
  • Quinine / pharmacology
  • Thiocyanates / pharmacology
  • Valinomycin / pharmacology

Substances

  • Thiocyanates
  • Valinomycin
  • Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone
  • Calcimycin
  • Potassium Chloride
  • Quinine
  • Potassium
  • potassium thiocyanate