Abstract
Inflammatory diseases typically display circadian variation in symptom severity. A new study in mice shows how a pulmonary epithelial cell clock controls neutrophil recruitment to the lungs and provides insight into interactions between local and systemic circadian clocks.
MeSH terms
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ARNTL Transcription Factors / immunology*
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Animals
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Chemokine CXCL5 / immunology*
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Circadian Clocks / immunology*
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Glucocorticoids / pharmacology*
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Humans
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Pneumonia, Pneumococcal / immunology*
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Streptococcus pneumoniae*
Substances
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ARNTL Transcription Factors
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Bmal1 protein, mouse
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Chemokine CXCL5
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Cxcl5 protein, mouse
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Glucocorticoids