Performance of amplicon-based next generation DNA sequencing for diagnostic gene mutation profiling in oncopathology

Cell Oncol (Dordr). 2014 Oct;37(5):353-61. doi: 10.1007/s13402-014-0196-2. Epub 2014 Sep 11.

Abstract

Purpose: Next generation DNA sequencing (NGS) holds promise for diagnostic applications, yet implementation in routine molecular pathology practice requires performance evaluation on DNA derived from routine formalin-fixed paraffin-embedded (FFPE) tissue specimens. The current study presents a comprehensive analysis of TruSeq Amplicon Cancer Panel-based NGS using a MiSeq Personal sequencer (TSACP-MiSeq-NGS) for somatic mutation profiling.

Methods: TSACP-MiSeq-NGS (testing 212 hotspot mutation amplicons of 48 genes) and a data analysis pipeline were evaluated in a retrospective learning/test set approach (n = 58/n = 45 FFPE-tumor DNA samples) against 'gold standard' high-resolution-melting (HRM)-sequencing for the genes KRAS, EGFR, BRAF and PIK3CA. Next, the performance of the validated test algorithm was assessed in an independent, prospective cohort of FFPE-tumor DNA samples (n = 75).

Results: In the learning set, a number of minimum parameter settings was defined to decide whether a FFPE-DNA sample is qualified for TSACP-MiSeq-NGS and for calling mutations. The resulting test algorithm revealed 82% (37/45) compliance to the quality criteria and 95% (35/37) concordant assay findings for KRAS, EGFR, BRAF and PIK3CA with HRM-sequencing (kappa = 0.92; 95% CI = 0.81-1.03) in the test set. Subsequent application of the validated test algorithm to the prospective cohort yielded a success rate of 84% (63/75), and a high concordance with HRM-sequencing (95% (60/63); kappa = 0.92; 95% CI = 0.84-1.01). TSACP-MiSeq-NGS detected 77 mutations in 29 additional genes.

Conclusion: TSACP-MiSeq-NGS is suitable for diagnostic gene mutation profiling in oncopathology.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biomarkers, Tumor / genetics
  • Class I Phosphatidylinositol 3-Kinases
  • DNA Mutational Analysis / methods*
  • DNA, Neoplasm / chemistry
  • DNA, Neoplasm / genetics*
  • ErbB Receptors / genetics
  • Formaldehyde / chemistry
  • HCT116 Cells
  • High-Throughput Nucleotide Sequencing / methods*
  • Humans
  • Mutation*
  • Neoplasms / diagnosis
  • Neoplasms / genetics*
  • Paraffin Embedding / methods
  • Phosphatidylinositol 3-Kinases / genetics
  • Polymerase Chain Reaction / methods
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins B-raf / genetics
  • Proto-Oncogene Proteins p21(ras)
  • Reproducibility of Results
  • Sensitivity and Specificity
  • Tissue Fixation / methods
  • ras Proteins / genetics

Substances

  • Biomarkers, Tumor
  • DNA, Neoplasm
  • KRAS protein, human
  • Proto-Oncogene Proteins
  • Formaldehyde
  • Phosphatidylinositol 3-Kinases
  • Class I Phosphatidylinositol 3-Kinases
  • PIK3CA protein, human
  • EGFR protein, human
  • ErbB Receptors
  • BRAF protein, human
  • Proto-Oncogene Proteins B-raf
  • Proto-Oncogene Proteins p21(ras)
  • ras Proteins