Monoaminergic regulation of the levels of beta-endorphin-like immunoreactivity (beta-ENDi) in rat hypothalamic nuclei

Neuropeptides. 1989 May-Jun;13(4):285-9. doi: 10.1016/0143-4179(89)90083-8.

Abstract

To examine the role of monoamines in regulating beta-endorphin levels in discrete brain nuclei, the levels of beta-endorphin-like immunoreactivity (beta-ENDi) were determined in the hypothalamic nuclei of rats 2 h after they were treated with monoaminergic drugs. Sulpiride decreased the levels of beta-ENDi in the nucleus paraventricularis, nucleus arcuatus and median eminence. Domperidone decreased the levels of beta-ENDi in the nucleus aracuatus and median eminence. L-DOPA increased the levels of beta-ENDi in the nucleus anterior hypothalami and median eminence. Phenylephrine or prazosine did not alter the levels of beta-ENDi. Yohimine decreased the levels of beta-ENDI in the nucleus anterior hypothalami. Isoproterenol increased the levels of beta-ENDi in the nucleus arcuatus, and propranolol reversed this effect. These results suggest that dopamine and noradrenaline (via beta-adrenoceptors) may regulate beta-endorphinergic neurons in the rat hypothalamus.

MeSH terms

  • Animals
  • Autonomic Agents / pharmacology*
  • Biogenic Monoamines / physiology*
  • Domperidone / pharmacology
  • Hypothalamus / analysis
  • Hypothalamus / drug effects*
  • Male
  • Radioimmunoassay
  • Rats
  • Rats, Inbred Strains
  • Receptors, Adrenergic / drug effects
  • Receptors, Adrenergic / physiology
  • Receptors, Dopamine / drug effects
  • Receptors, Dopamine / physiology
  • Sulpiride / pharmacology
  • beta-Endorphin / analysis*
  • beta-Endorphin / physiology

Substances

  • Autonomic Agents
  • Biogenic Monoamines
  • Receptors, Adrenergic
  • Receptors, Dopamine
  • Domperidone
  • beta-Endorphin
  • Sulpiride