Abstract
RunX2 has been identified to crucially regulate the osteolysis in giant cell tumor of bone. MiR-30a is an intronic miRNA identified as tumor suppressor, but little is known about its role in giant tumor cell of bone. In our research, we reported miR-30a was down-regulated in GCT whereas RunX2 was highly expressed. Further research proved that miR-30a can regulate the expression of RunX2 by binding to its 3'-UTR, which influence the osteoclast differentiation and osteolysis formation. Thus, these results suggest that miR-30a could directly target RunX2 and participate in osteolysis in GCT.
Keywords:
Giant cell tumor of bone; Osteoclast differentiation; RunX2; miR-30a-5p.
Copyright © 2014 Elsevier Inc. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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3' Untranslated Regions
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Animals
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Base Sequence
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Bone Neoplasms / genetics*
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Bone Neoplasms / metabolism
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Bone Neoplasms / pathology*
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Core Binding Factor Alpha 1 Subunit / genetics*
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Down-Regulation
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Gene Expression Regulation, Neoplastic
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Genes, Tumor Suppressor
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Giant Cell Tumor of Bone / genetics*
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Giant Cell Tumor of Bone / metabolism
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Giant Cell Tumor of Bone / pathology*
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Humans
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Mice
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Mice, Inbred C57BL
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MicroRNAs / genetics*
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MicroRNAs / metabolism
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Molecular Sequence Data
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Osteoclasts / metabolism
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Osteoclasts / pathology
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Osteolysis / genetics*
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Osteolysis / pathology
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Osteolysis / prevention & control*
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RNA, Neoplasm / genetics
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RNA, Neoplasm / metabolism
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Tumor Cells, Cultured
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Up-Regulation
Substances
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3' Untranslated Regions
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Core Binding Factor Alpha 1 Subunit
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MIRN30b microRNA, human
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MicroRNAs
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RNA, Neoplasm
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RUNX2 protein, human