Lymphocyte migration into atherosclerotic plaque

Arterioscler Thromb Vasc Biol. 2015 Jan;35(1):40-9. doi: 10.1161/ATVBAHA.114.303227. Epub 2014 Oct 9.

Abstract

Adaptive immunity is involved in the pathogenesis of atherosclerosis, but the recruitment of T and B lymphocytes to atherosclerotic lesions is not as well studied as that of monocytes. In this review, we summarize the current understanding of the role of lymphocyte subsets in the pathogenesis of atherosclerosis and discuss chemokines and chemokine receptors involved in lymphocyte homing to atherosclerotic lesions. We review evidence for involvement of the chemokines CCL5, CCL19, CCL21, CXCL10, and CXCL16 and macrophage migration inhibitory factor in lymphocyte homing in atherosclerosis. Also, we review the role of their receptors CCR5, CCR6, CCR7, CXCR3, CXCR6, and CXCR2/CXCR4 and the role of the L-selectin in mouse models of atherosclerosis.

Keywords: CC chemokine receptor; atherosclerosis; lymphocytes.

Publication types

  • Review

MeSH terms

  • Animals
  • Atherosclerosis / immunology*
  • Atherosclerosis / metabolism
  • Atherosclerosis / pathology
  • Blood Vessels / immunology*
  • Blood Vessels / metabolism
  • Blood Vessels / pathology
  • Chemokines / metabolism
  • Chemotaxis, Leukocyte*
  • Disease Models, Animal
  • Humans
  • L-Selectin / metabolism
  • Lymphocyte Subsets / immunology*
  • Lymphocyte Subsets / metabolism
  • Lymphocyte Subsets / pathology
  • Macrophage Migration-Inhibitory Factors / metabolism
  • Mice
  • Plaque, Atherosclerotic*
  • Receptors, Chemokine / metabolism
  • Signal Transduction

Substances

  • Chemokines
  • Macrophage Migration-Inhibitory Factors
  • Receptors, Chemokine
  • L-Selectin