Evidence for testicular impairment after long-term treatment with a luteinizing hormone-releasing hormone agonist in elderly men

J Urol. 1989 Nov;142(5):1235-8. doi: 10.1016/s0022-5347(17)39042-0.

Abstract

Testicular responsiveness to 5,000 IU of human chorionic gonadotropin was evaluated in 14 patients with prostate cancer who were being treated with a slow-release luteinizing hormone-releasing hormone agonist for a median of 21 months. Serum testosterone response to human chorionic gonadotropin was markedly reduced in most patients, with the median level increasing from 0.25 to 1.65 nmol. per l. A second human chorionic gonadotropin test was repeated later in 5 patients who had been off treatment for a median of 6 months. Median serum testosterone levels increased to a maximum of 2.6 nmol. per l. compared to 28.2 nmol. per l. in an age-matched control group (p equals 0.008). Therefore, we conclude that long-term treatment with luteinizing hormone-releasing hormone agonists in elderly men leads to gonadal impairment that may not be as reversible as generally suggested.

MeSH terms

  • 17-alpha-Hydroxyprogesterone
  • Aged
  • Antineoplastic Agents / adverse effects*
  • Antineoplastic Agents / therapeutic use
  • Chorionic Gonadotropin
  • Follicle Stimulating Hormone / metabolism
  • Gonadotropin-Releasing Hormone / adverse effects
  • Gonadotropin-Releasing Hormone / therapeutic use
  • Humans
  • Hydroxyprogesterones / metabolism
  • Luteinizing Hormone / metabolism
  • Male
  • Middle Aged
  • Prostatic Neoplasms / drug therapy
  • Prostatic Neoplasms / metabolism
  • Testis / drug effects*
  • Testis / metabolism
  • Testosterone / metabolism
  • Triptorelin Pamoate

Substances

  • Antineoplastic Agents
  • Chorionic Gonadotropin
  • Hydroxyprogesterones
  • Triptorelin Pamoate
  • Gonadotropin-Releasing Hormone
  • Testosterone
  • 17-alpha-Hydroxyprogesterone
  • Luteinizing Hormone
  • Follicle Stimulating Hormone