The expression of cAMP-dependent protein kinase subunits is differentially regulated during liver regeneration

J Biol Chem. 1989 Mar 15;264(8):4374-82.

Abstract

The levels of the regulatory (RI and RII) and catalytic (C) components of cAMP-dependent protein kinase and of their messages were studied during the first 36 h of liver regeneration after 70% hepatectomy. Both RI alpha mRNA and RII alpha mRNA started to increase 4 h after the resection, reaching peak levels after 9 h. RI mRNA decreased abruptly 9-12 h after resection, whereas RII mRNA stayed elevated. C alpha mRNA was rather constant during the period of study. In accordance with the mRNA data the level of C was constant while RI and RII increased during the prereplicative phase of liver regeneration. RI increased rapidly when its message became elevated. RII, however, increased noticeably only 6-8 h after its mRNA had become elevated. The increased expression of R led to a disproportion between R and C that was most pronounced 14 h after resection, i.e. coinciding with the prereplicative cAMP burst. The increased R/C ratio at that time of regeneration diminished the concentration of active C subunit during the cAMP burst. In that way the otherwise inhibitory effect of high concentrations of active C on the DNA replication may have been decreased. The fractional saturation of RI and RII by endogenous cAMP fluctuated in parallel as a function of liver cAMP levels, although there was a tendency that RI was more highly saturated than RII at high concentrations of cAMP.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cyclic AMP / metabolism
  • Cyclic AMP / pharmacology*
  • Cytosol / enzymology
  • DNA Replication
  • Gene Expression Regulation*
  • Hepatectomy
  • Kinetics
  • Liver / enzymology*
  • Liver Regeneration*
  • Macromolecular Substances
  • Male
  • Nucleic Acid Hybridization
  • Protein Kinases / biosynthesis
  • Protein Kinases / genetics*
  • RNA, Messenger / biosynthesis
  • Rats
  • Rats, Inbred Strains

Substances

  • Macromolecular Substances
  • RNA, Messenger
  • Cyclic AMP
  • Protein Kinases