Effective suppression of the Kirsten rat sarcoma viral oncogene in pancreatic tumor cells via targeted small interfering RNA delivery using nanoparticles

Pancreas. 2015 Mar;44(2):250-9. doi: 10.1097/MPA.0000000000000241.

Abstract

Objectives: The objective of this study was to establish an efficient carrier for small interfering RNA (siRNA) delivery targeting pancreatic tumor cells.

Methods: A copolymer consisting of a single-chain variable fragment targeted to human CD44 variant 6 (scFv(CD44v6)) functional group conjugated to polyethylene glycol-poly-L-lysine was synthesized and assembled into micelles encapsulating the siRNAs. Flow cytometry and Western blot assays were performed to evaluate the transfection efficiency and gene-silencing effect of the siRNAs. Afterward, (4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetrazolium bromide, Transwell, soft agar colony formation, and enzyme-linked immunosorbent assays were performed to evaluate the biological functions of PANC-1 cells after Kirsten rat sarcoma viral oncogene knockdown. In vivo assays were performed using a BALB/c (nu/nu) mouse model subcutaneously injected with PANC-1 xenografts. Real-time in vivo fluorescence imaging was used to monitor the tumor homing of the nanoparticles.

Results: The scFv(CD44v6) enabled more efficient delivery of siRNAs and exhibited enhanced gene silencing compared with nontargeted nanoparticles. Furthermore, targeted delivery of the siRNAs induced a potent inhibitory effect on cell proliferation, colony formation, invasion, and vascular endothelial growth factor production. The animal assays revealed that single-chain variable fragment nanoparticles accumulated in the tumor tissue and enhanced the inhibition of tumor growth in vivo.

Conclusions: The scFv(CD44v6)-conjugated nanocarriers provide a highly efficient and safe platform for systemic gene therapy for pancreatic cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Movement
  • Cell Proliferation
  • Female
  • Gene Expression Regulation, Neoplastic
  • Gene Knockdown Techniques
  • Genetic Therapy / methods*
  • Humans
  • Hyaluronan Receptors / immunology
  • Hyaluronan Receptors / metabolism
  • Lysine / analogs & derivatives
  • Lysine / chemistry
  • Mice, Inbred BALB C
  • Mice, Nude
  • Micelles
  • Nanomedicine
  • Nanoparticles*
  • Neoplasm Invasiveness
  • Pancreatic Neoplasms / genetics
  • Pancreatic Neoplasms / immunology
  • Pancreatic Neoplasms / metabolism
  • Pancreatic Neoplasms / pathology
  • Pancreatic Neoplasms / therapy*
  • Polyethylene Glycols / chemistry
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins / metabolism
  • Proto-Oncogene Proteins p21(ras)
  • RNA Interference*
  • RNA, Small Interfering / genetics*
  • RNA, Small Interfering / metabolism
  • Single-Chain Antibodies / metabolism
  • Time Factors
  • Transfection / methods*
  • Tumor Burden
  • U937 Cells
  • Vascular Endothelial Growth Factor A / metabolism
  • Xenograft Model Antitumor Assays
  • ras Proteins / genetics*
  • ras Proteins / metabolism

Substances

  • CD44 protein, human
  • Hyaluronan Receptors
  • KRAS protein, human
  • Micelles
  • Proto-Oncogene Proteins
  • RNA, Small Interfering
  • Single-Chain Antibodies
  • VEGFA protein, human
  • Vascular Endothelial Growth Factor A
  • poly(ethylene glycol) lysine copolymer
  • Polyethylene Glycols
  • Proto-Oncogene Proteins p21(ras)
  • ras Proteins
  • Lysine