Mouse dead end 1-β interacts with c-Jun and stimulates activator protein 1 transactivation

Mol Med Rep. 2015 Mar;11(3):1701-7. doi: 10.3892/mmr.2014.2950. Epub 2014 Nov 14.

Abstract

Dead end 1 (DND1), important for maintaining the viability of primordial germ cells, is the first protein containing an RNA recognition motif that has been directly implicated as a heritable cause of spontaneous tumorigenesis. In the present study, c-Jun was identified through yeast two-hybrid screening of a 10.5-day old mouse embryo cDNA library as one of the proteins which interact with DND1-β. The interaction between DND1-β and c-Jun was demonstrated to occur by glutathione S‑transferase pull‑down and co-immunoprecipitation. Using confocal microscopy, DND1-β was found to be specifically expressed in GC-1 spermatogonia cells, mainly in the nuclei. When transfected into GC-1 cells, DND1-β and c-Jun were demonstrated to be co-localized principally in the nuclei. Furthermore, in a dual luciferase reporter assay, the transcriptional activity of activator protein 1 was demonstrated to be significantly increased by co-transfection with DND1-β and c-Jun plasmids in GC-1 cells. The identification and confirmation of an additional protein interacting with DND1-β facilitates the investigation of the functions and molecular mechanisms of DND1.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • Gene Expression Regulation
  • Mice
  • Neoplasm Proteins / metabolism*
  • Protein Binding
  • Protein Interaction Mapping
  • Proto-Oncogene Proteins c-jun / metabolism*
  • Transcription Factor AP-1 / metabolism*
  • Transcriptional Activation*
  • Two-Hybrid System Techniques

Substances

  • Dnd1 protein, mouse
  • Neoplasm Proteins
  • Proto-Oncogene Proteins c-jun
  • Transcription Factor AP-1