The effect of the long-acting somatostatin analogue SMS 201-995 on ACTH secretion in Nelson's syndrome and Cushing's disease

Acta Endocrinol (Copenh). 1989 Jun;120(6):760-6. doi: 10.1530/acta.0.1200760.

Abstract

Chronic therapy of a patient with Nelson's syndrome for 2 years with 300 micrograms SMS 201-995 per day resulted in a significant decrease in circulating ACTH levels, normalization of the visual field defect and of loss of visual acuity of one eye, and stabilization of tumour growth, without radiological evidence of shrinkage of the pituitary tumour. In two other patients with Nelson's syndrome, SMS 201-995 acutely inhibited circulating ACTH levels. This effect could be shown best if cortisol replacement was temporarily withheld. SMS 201-995 did not affect plasma ACTH and cortisol levels in three patients with untreated Cushing's disease.

Publication types

  • Case Reports
  • Clinical Trial

MeSH terms

  • Adrenocorticotropic Hormone / metabolism*
  • Adult
  • Clinical Trials as Topic
  • Cushing Syndrome / blood
  • Cushing Syndrome / drug therapy*
  • Cushing Syndrome / physiopathology
  • Delayed-Action Preparations
  • Female
  • Humans
  • Male
  • Nelson Syndrome / blood
  • Nelson Syndrome / drug therapy*
  • Nelson Syndrome / physiopathology
  • Octreotide / administration & dosage*
  • Pituitary Neoplasms / drug therapy*
  • Time Factors
  • Visual Acuity / drug effects

Substances

  • Delayed-Action Preparations
  • Adrenocorticotropic Hormone
  • Octreotide