A type 2A protein phosphatase dephosphorylates the elongation factor 2 and is stimulated by the phorbol ester TPA in mouse epidermis in vivo

FEBS Lett. 1989 Nov 6;257(2):357-60. doi: 10.1016/0014-5793(89)81571-6.

Abstract

Mouse epidermal cytosol contains a protein phosphatase with Mr 38,000, which dephosphorylates the elongation factor 2 (EF-2) of protein biosynthesis and is stimulated after topical application of TPA to mouse skin [(1988) Biochem. Biophys. Res. Commun. 153, 1129-1135]. Dephosphorylation of EF-2 by this phosphatase is inhibited by okadaic acid at concentrations as low as 10(-8) M, but not by heparin up to concentrations of 600.micrograms/ml. The catalytic subunit of protein phosphatase 2A (PP2Ac) with EF-2 as a substrate exhibits the same sensitivity towards okadaic acid and insensitivity towards heparin as the EF-2 phosphatase of epidermal cytosol. The catalytic subunit of protein phosphatase 1 (PP1c) is strongly suppressed by heparin and less sensitive towards okadaic acid than PP2Ac. PP2Ac is around 50 times more efficient in dephosphorylating EF-2 than PP1c. These data indicate that the TPA-stimulated EF-2 phosphatase in epidermal cytosol is a type 2A protein phosphatase.

MeSH terms

  • Animals
  • Cytosol / enzymology
  • Enzyme Activation / drug effects
  • Epidermis / metabolism*
  • Ethers, Cyclic / pharmacology
  • Heparin / pharmacology
  • Mice
  • Molecular Weight
  • Okadaic Acid
  • Peptide Elongation Factor 2
  • Peptide Elongation Factors / metabolism*
  • Phosphoprotein Phosphatases / metabolism*
  • Protein Phosphatase 1
  • Protein Phosphatase 2
  • Tetradecanoylphorbol Acetate / pharmacology*

Substances

  • Ethers, Cyclic
  • Peptide Elongation Factor 2
  • Peptide Elongation Factors
  • Okadaic Acid
  • Heparin
  • Phosphoprotein Phosphatases
  • Protein Phosphatase 1
  • Protein Phosphatase 2
  • Tetradecanoylphorbol Acetate