[Effects of intrahippocampal delta-receptors on inhibition of electroconvulsive shock by electro-acupuncture]

Zhongguo Yao Li Xue Bao. 1989 May;10(3):197-201.
[Article in Chinese]

Abstract

The relationship between the delta-receptors of the hippocampus and electro-acupuncture (EA) anticonvulsions was analyzed by radioimmunoassay and microinjection. The content of leu-enkephalin-like immunoreactive substance (LEK-LIS) in perfusate from the hippocampus increased after repeated electroconvulsive shock (ECS), while the content of LEK-LIS was not changed after single ECS. EA inhibited epileptiform spikes induced by ECS, and the content of LEK-LIS decreased markedly after EA (30 +/- 9 pg/0.1 ml lower than that before EA). Leu-enkephalin (LEK), an agonist of the delta-receptors, 10 micrograms injected into the hippocampus produced epileptiform spikes. However, ICI 174,864 (4 micrograms), a specific antagonist of the delta-receptors, suppressed convulsions significantly. The results suggest that the delta-receptors of the hippocampus participate in EA anticonvulsions.

Publication types

  • English Abstract

MeSH terms

  • Animals
  • Electroacupuncture*
  • Electroshock
  • Enkephalin, Leucine / analogs & derivatives
  • Enkephalin, Leucine / metabolism
  • Enkephalin, Leucine / pharmacology
  • Female
  • Hippocampus / physiology*
  • Male
  • Narcotic Antagonists
  • Rats
  • Receptors, Opioid / physiology*
  • Receptors, Opioid, delta

Substances

  • Narcotic Antagonists
  • Receptors, Opioid
  • Receptors, Opioid, delta
  • Enkephalin, Leucine
  • N,N-diallyl-tyrosyl-alpha-aminoisobutyric acid-phenylalanyl-leucine