[Mechanisms of sorafenib induced NB4 cell apoptosis]

Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2015 Feb;23(1):77-82. doi: 10.7534/j.issn.1009-2137.2015.01.015.
[Article in Chinese]

Abstract

Objective: To investigate the effects of sorafenib on human acute promyelocytic leukemia cell NB4 and its mechanism.

Methods: The human acute promyelocytic leukemia cell NB4 was treated with different concentrations (0, 1.5, 3, 6 and 12 µmol/L) of sorafenib, the proliferation inhibitory rate of NB4 cells was assayed by MTT, the apoptosis of NB4 was determined with flow-cytomatry after treatment; after extraction of total protein, the Western blot was performed to determine the expressions of apoptosis-relatived molecules Caspase-3, Caspase-8 and MCL-1. The mRNA expressions of Caspase-3, Caspase-8 and MCL-1 were determined by RT-PCR.

Results: As compared with the control group, the proliferation of NB4 significantly decreased after treatment with different concentrations of sorafenib. The sorafenib significantly induced the apopotosis of NB4 cells in time- and dose-dependent manners. Furthermore, sorafenib treatment resulted in the obvious increase of the Caspase-3 and Caspase-8 protein and mRNA expressions, and down-regulated the MCL-1 protein and mRNA expressions in NB4 cells.

Conclusion: Sorafenib can inhibit proliferation and induce apopotosis of human acute promyelocytic leukemia cell NB4 through the expression of Caspase-3 and Caspase-8, and down-regulation of the expression of MCL-1.

MeSH terms

  • Antineoplastic Agents
  • Apoptosis*
  • Caspase 3
  • Caspase 8
  • Cell Line, Tumor
  • Down-Regulation
  • Humans
  • Leukemia, Promyelocytic, Acute
  • Niacinamide / analogs & derivatives
  • Phenylurea Compounds
  • Sorafenib
  • T-Lymphocytes, Helper-Inducer

Substances

  • Antineoplastic Agents
  • Phenylurea Compounds
  • Niacinamide
  • Sorafenib
  • CASP3 protein, human
  • CASP8 protein, human
  • Caspase 3
  • Caspase 8