Activin A can induce definitive endoderm differentiation from human parthenogenetic embryonic stem cells

Biotechnol Lett. 2015 Aug;37(8):1711-7. doi: 10.1007/s10529-015-1829-x. Epub 2015 Apr 8.

Abstract

Objectives: As activin/nodal signaling plays a key role in definitive endoderm (DE) differentiation, we have explored activin A-induced differentiation of DE from human parthenogenetic embryonic stem cells (hPESCs).

Results: Administration of 5 ng activin A/ml had no effect on the expression of markers of DE differentiation. However, higher concentrations of activin A (50 and 100 ng/ml) upregulated Sox17 and Cxcr4, as well upregulating the mesendodermal precursor marker, Brachyury.

Conclusions: These findings demonstrate that low dose activin A can maintain the undifferentiated potency of hPESCs, whereas higher doses induce DE differentiation; 50 ng/ml is the optimal concentration for inducing DE from hPESCs.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Activins / metabolism*
  • Biomarkers / analysis
  • Cell Differentiation / drug effects*
  • Embryonic Stem Cells / drug effects*
  • Endoderm / chemistry
  • Endoderm / growth & development*
  • Fetal Proteins / analysis
  • Humans
  • Receptors, CXCR4 / analysis
  • SOXF Transcription Factors / analysis
  • T-Box Domain Proteins / analysis

Substances

  • Biomarkers
  • CXCR4 protein, human
  • Fetal Proteins
  • Receptors, CXCR4
  • SOX17 protein, human
  • SOXF Transcription Factors
  • T-Box Domain Proteins
  • activin A
  • Activins
  • Brachyury protein