Abstract
The in vivo administration of ethinylestradiol to female rats causes dose-dependent decreases in tri-, di- and monoacylglycerol lipase activities in post-heparin heart effluents; monoacylglycerol lipase is less sensitive to estrogen than the other two.
MeSH terms
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Animals
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Body Weight
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Carboxylic Ester Hydrolases / antagonists & inhibitors*
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Dose-Response Relationship, Drug
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Ethinyl Estradiol / pharmacology*
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Female
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Heparin / pharmacology
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Lipoprotein Lipase / antagonists & inhibitors*
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Monoacylglycerol Lipases / antagonists & inhibitors*
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Myocardium / enzymology*
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Norethindrone / pharmacology
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Perfusion
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Rats
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Rats, Inbred Strains
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Regression Analysis
Substances
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Ethinyl Estradiol
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Heparin
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Carboxylic Ester Hydrolases
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Monoacylglycerol Lipases
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Lipoprotein Lipase
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Norethindrone