Cancer metabolism: A waste of insulin interference

Nature. 2015 May 28;521(7553):430-1. doi: 10.1038/521430a.
No abstract available

MeSH terms

  • Adaptor Proteins, Signal Transducing / metabolism
  • Animals
  • Cachexia / etiology
  • Cachexia / metabolism*
  • Disease Models, Animal
  • Drosophila Proteins / antagonists & inhibitors
  • Drosophila Proteins / metabolism
  • Drosophila melanogaster / metabolism
  • Humans
  • Insulin / metabolism*
  • Insulin Resistance
  • Insulin-Like Growth Factor Binding Proteins / antagonists & inhibitors
  • Insulin-Like Growth Factor Binding Proteins / metabolism
  • Insulin-Like Growth Factor I / metabolism
  • Mice
  • Neoplasms / complications
  • Neoplasms / genetics
  • Neoplasms / metabolism*
  • Nuclear Proteins / metabolism
  • Phosphoproteins / metabolism
  • Signal Transduction
  • Trans-Activators / metabolism
  • Transcription Factors
  • YAP-Signaling Proteins

Substances

  • Adaptor Proteins, Signal Transducing
  • Drosophila Proteins
  • ImpL2 protein, Drosophila
  • Insulin
  • Insulin-Like Growth Factor Binding Proteins
  • Nuclear Proteins
  • Phosphoproteins
  • Trans-Activators
  • Transcription Factors
  • YAP-Signaling Proteins
  • YAP1 protein, human
  • Yki protein, Drosophila
  • Insulin-Like Growth Factor I