Suppression of multidrug resistance by rosiglitazone treatment in human ovarian cancer cells through downregulation of FZD1 and MDR1 genes

Anticancer Drugs. 2015 Aug;26(7):706-15. doi: 10.1097/CAD.0000000000000236.

Abstract

Multidrug resistance (MDR) is a major obstacle in the successful treatment of ovarian cancer. One of the most common causes of MDR is overexpression of P-glycoprotein (P-gp), encoded by the MDR1 gene. The MDR1 gene is a direct target of the Wnt/β-catenin signaling pathway, which plays an important role in ovarian cancer. Peroxisome proliferator-activated receptor γ (PPARγ) ligands have been found to protect against development of cancer through the Wnt/β-catenin pathway. To investigate the effect of PPARγ ligands on MDR1/P-gp expression, we treated a MDR ovarian cancer cell subline, A2780/Taxol, with different concentrations of rosiglitazone (Rosi), a member of the synthetic PPARγ ligands. Rosi downregulated FZD1 and MDR1/P-gp expression in a concentration-dependent manner. In addition, nuclear β-catenin levels and its transcriptional activity decreased significantly. In conclusion, Rosi may reverse MDR of ovarian cancer cells by downregulating the Wnt/β-catenin pathway with the suppression of FZD1.

MeSH terms

  • ATP Binding Cassette Transporter, Subfamily B, Member 1 / antagonists & inhibitors
  • ATP Binding Cassette Transporter, Subfamily B, Member 1 / metabolism
  • Active Transport, Cell Nucleus
  • Anilides / pharmacology
  • Antineoplastic Agents / pharmacology*
  • Cell Line, Tumor
  • Cell Nucleus / metabolism
  • Cisplatin / pharmacology
  • Down-Regulation
  • Doxorubicin / pharmacology
  • Drug Resistance, Multiple / drug effects
  • Drug Resistance, Neoplasm / drug effects*
  • Female
  • Fluorouracil / pharmacology
  • Frizzled Receptors / antagonists & inhibitors
  • Frizzled Receptors / metabolism
  • Humans
  • Ovarian Neoplasms
  • PPAR gamma / agonists*
  • PPAR gamma / antagonists & inhibitors
  • PPAR gamma / metabolism
  • Paclitaxel / pharmacology
  • Rosiglitazone
  • Signal Transduction
  • Thiazolidinediones / pharmacology*
  • Transcription, Genetic
  • Wnt Proteins / metabolism
  • beta Catenin / genetics
  • beta Catenin / metabolism

Substances

  • 2-chloro-5-nitrobenzanilide
  • ATP Binding Cassette Transporter, Subfamily B, Member 1
  • Anilides
  • Antineoplastic Agents
  • FZD1 protein, human
  • Frizzled Receptors
  • PPAR gamma
  • Thiazolidinediones
  • Wnt Proteins
  • beta Catenin
  • Rosiglitazone
  • Doxorubicin
  • Paclitaxel
  • Cisplatin
  • Fluorouracil