Advances in EGFR as a Predictive Marker in Lung Adenocarcinoma

Cancer Control. 2015 Apr;22(2):193-9. doi: 10.1177/107327481502200210.

Abstract

Background: Worldwide, lung cancer is the most common cause of mortality. Toxins from tobacco smoke are known to increase the risk of lung cancer; however, up to 15% of lung cancer-related deaths in men and up to 50% of lung cancer-related deaths in women occur in people who do not smoke. Despite the fact that chemotherapy generally provides a survival benefit for non-small-cell lung cancer, not every patient will respond to therapy and many experience therapy-related adverse events. Thus, predictive markers are used to determine which patients are more likely to respond to a given regimen.

Methods: We reviewed the current medical literature in English relating to predictive markers that may be positive, such as the presence of an activating EGFR mutation.

Results: The advances in using EGFR as a molecular predictive marker were summarized. This biomarker influences therapeutic response in patients with lung adenocarcinoma. Clinical evidence supporting its value is also reviewed.

Conclusions: The use of EGFR as a predictive factor in lung adenocarcinoma may help target therapy to individual tumors to achieve the best likelihood for long-term survival and to avoid adverse events from medications unlikely to be effective.

Publication types

  • Review

MeSH terms

  • Adenocarcinoma / genetics*
  • Adenocarcinoma / pathology
  • Adenocarcinoma of Lung
  • Amino Acid Substitution
  • Biomarkers, Tumor
  • Carcinoma, Non-Small-Cell Lung / genetics*
  • Carcinoma, Non-Small-Cell Lung / pathology
  • Chromosome Deletion
  • Drug Resistance, Neoplasm / genetics
  • ErbB Receptors / genetics*
  • Exons / genetics
  • Humans
  • Lung Neoplasms / genetics*
  • Lung Neoplasms / pathology
  • Mutation
  • Prognosis
  • Protein-Tyrosine Kinases / antagonists & inhibitors*

Substances

  • Biomarkers, Tumor
  • ErbB Receptors
  • Protein-Tyrosine Kinases