Cytokine-Mediated Loss of Blood Dendritic Cells During Epstein-Barr Virus-Associated Acute Infectious Mononucleosis: Implication for Immune Dysregulation

J Infect Dis. 2015 Dec 15;212(12):1957-61. doi: 10.1093/infdis/jiv340. Epub 2015 Jun 16.

Abstract

Acute infectious mononucleosis (IM) is associated with altered expression of inflammatory cytokines and disturbed T-cell homeostasis, however, the precise mechanism of this immune dysregulation remains unresolved. In the current study we demonstrated a significant loss of circulating myeloid and plasmacytoid dendritic cells (DCs) during acute IM, a loss correlated with the severity of clinical symptoms. In vitro exposure of blood DCs to acute IM plasma resulted in loss of plasmacytoid DCs, and further studies with individual cytokines showed that exposure to interleukin 10 could replicate this effect. Our data provide important mechanistic insight into dysregulated immune homeostasis during acute IM.

Keywords: acute infection; anti-viral immunity; cytokines; dendritic cells; herpesvirus; immune dysregulation; immune regulation; virus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Blood Cells / drug effects*
  • Cell Survival / drug effects
  • Dendritic Cells / immunology*
  • Humans
  • Immune Tolerance*
  • Infectious Mononucleosis / immunology
  • Infectious Mononucleosis / pathology*
  • Interleukin-10 / blood*
  • Interleukin-10 / metabolism
  • Young Adult

Substances

  • IL10 protein, human
  • Interleukin-10