Characterization of Clinical Cases of Collecting Duct Carcinoma of the Kidney Assessed by Comprehensive Genomic Profiling

Eur Urol. 2016 Sep;70(3):516-21. doi: 10.1016/j.eururo.2015.06.019. Epub 2015 Jul 3.

Abstract

Background: Collecting duct carcinoma (CDC) is a rare type of renal cell carcinoma (RCC) originating from the renal medulla. Clinical outcomes are poor, and there are no consensus guidelines to guide therapy.

Objective: To determine genomic alterations (GAs) in a series of patients with locally advanced or metastatic CDC for whom genomic profiling was performed during the course of clinical care.

Design, setting, and participants: Formalin-fixed, paraffin-embedded blocks or slides were obtained for 17 patients with CDC. DNA was extracted and comprehensive genomic profiling was performed in a laboratory certified under the Clinical Laboratory Improvement Amendments.

Outcome measurements and statistical analysis: Bayesian algorithms and local alignment algorithms were used to detect substitutions and insertions/deletions, respectively. A comparison to normal control samples was used to detect copy number alterations. Clinically relevant GAs (CRGAs) were defined as those linked to approved or investigational targeted therapies.

Results and limitations: The median age in the cohort was 53 yr (range 26-73), and 14 primary tumors and three metastatic sites assessed. A total of 36 GAs were detected in this series of patients, with an average of 2.1 GAs per case. The most common GAs were in NF2 (5/17, 29%), SETD2 (4/17, 24%), SMARCB1 (3/17, 18%), and CDKN2A (2/17, 12%). Of nine cases assessed for FH GAs, two patients had FH homozygous loss. A limitation is that targeted interrogation of genes known to be implicated in other cancers was performed, so mutations outside of these cannot be excluded.

Conclusions: Recurrent CRGAs were detected in this series of CDC cases and suggest a possible benefit from targeted therapy. In particular, mTOR inhibitors may be of interest in patients with NF2 alterations. Alterations in FH and SMARCB1 also occurred in a mutually exclusive manner to NF2 alterations.

Patient summary: This report provides important genomic insights into collecting duct carcinoma, a rare type of renal cell carcinoma with a very aggressive course. These insights could further rationalize the use of targeted therapies for rare tumors according to the individual genomic alterations harbored.

Keywords: Collecting duct carcinoma; Genomic profiling; Medullary carcinoma; NF2.

MeSH terms

  • Adult
  • Aged
  • Carcinoma, Renal Cell / genetics*
  • Carcinoma, Renal Cell / secondary
  • Class I Phosphatidylinositol 3-Kinases / genetics
  • DNA (Cytosine-5-)-Methyltransferases / genetics
  • DNA Copy Number Variations
  • DNA Methyltransferase 3A
  • F-Box-WD Repeat-Containing Protein 7 / genetics
  • Female
  • Fumarate Hydratase / genetics
  • Genes, Neurofibromatosis 2
  • Genes, p16
  • Histone-Lysine N-Methyltransferase / genetics
  • Humans
  • INDEL Mutation
  • Kidney Neoplasms / genetics*
  • Kidney Neoplasms / pathology
  • Kidney Tubules, Collecting
  • Male
  • Middle Aged
  • Phosphatidylinositol 3-Kinases / genetics
  • Proto-Oncogene Proteins p21(ras) / genetics
  • SMARCB1 Protein / genetics
  • Transcriptome
  • Tumor Suppressor Proteins / genetics
  • Ubiquitin Thiolesterase / genetics
  • Von Hippel-Lindau Tumor Suppressor Protein / genetics

Substances

  • BAP1 protein, human
  • DNMT3A protein, human
  • F-Box-WD Repeat-Containing Protein 7
  • FBXW7 protein, human
  • SMARCB1 Protein
  • SMARCB1 protein, human
  • Tumor Suppressor Proteins
  • DNA (Cytosine-5-)-Methyltransferases
  • DNA Methyltransferase 3A
  • Histone-Lysine N-Methyltransferase
  • SETD2 protein, human
  • Von Hippel-Lindau Tumor Suppressor Protein
  • phosphoinositol-3 kinase regulatory subunit 2, human
  • Class I Phosphatidylinositol 3-Kinases
  • PIK3CA protein, human
  • Ubiquitin Thiolesterase
  • HRAS protein, human
  • Proto-Oncogene Proteins p21(ras)
  • Fumarate Hydratase
  • VHL protein, human