Prevalence and phenotypes of congenital myopathy due to α-actin 1 gene mutations

Muscle Nerve. 2016 Mar;53(3):388-93. doi: 10.1002/mus.24765. Epub 2015 Aug 13.

Abstract

Introduction: Congenital myopathy due to mutations in the α-actin 1 gene (ACTA1) was identified in 1999, but knowledge of prevalence and phenotype in patients who survive 5 years is lacking.

Methods: A national cohort of 91 patients aged ≥5 years and diagnosed with congenital myopathy was assessed for ACTA1 mutations and investigated clinically.

Results: Four patients with ACTA1 mutations were identified, yielding a prevalence of 4.4%. Patients were 10-23 years of age, and all but 1 were ambulatory. Vital capacity ranged from 47% to 70% predicted, and 1 patient needed nocturnal bi-level positive airway pressure. Limb flexor/extensor muscles and upper and lower extremities were affected equally. Pronounced neck flexor weakness was noted.

Conclusions: Congenital myopathy caused by ACTA1 mutations is fatal in infancy in most cases. This study shows that the prevalence of α-actin myopathy in older patients with congenital myopathy is not negligible and that phenotypes can be quite mild.

Keywords: ACTA1; congenital myopathy; nemaline myopathy; phenotype; prevalence.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / genetics*
  • Adolescent
  • Child
  • Creatine Kinase / blood
  • Denmark
  • Female
  • Genetic Predisposition to Disease / genetics*
  • Humans
  • Male
  • Muscle, Skeletal / metabolism
  • Muscle, Skeletal / pathology
  • Mutation / genetics*
  • Myotonia Congenita / epidemiology*
  • Myotonia Congenita / genetics*
  • Myotonia Congenita / physiopathology
  • Phenotype
  • Prevalence
  • Young Adult

Substances

  • Actins
  • Creatine Kinase